TORRANCE MEMORIAL MEDICAL CENTER ****************************************************************** FOLLOWING CARDIAC ARREST



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TORRANCE MEMORIAL MEDICAL CENTER ****************************************************************** Department: Policy/Procedure: PATIENT CARE ****************************************************************** Policy: 1. This protocol outlines the approach for instituting immediate mild hypothermia (core temperature of 33-34 degrees Celsius) in patients presenting in coma following cardiac arrest (outof-hospital and in-hospital. Studies have shown that implementation of mild therapeutic hypothermia seems to improve survival and neurological outcome after cardiac arrest. 2. A physician s order is required to initiate this protocol. 3. This procedure may be performed in the Emergency Department, ICCU, or Cath Lab under the direction of the Emergency Department Physician or Cardiologist, and Intensivist. 4. Therapeutic hypothermia will be induced for up to 24 hours, and then the patient will be slowly rewarmed to bring safely back to normothermia. 5. The goal is to start cooling as soon as possible without delay. Code 90 patients will go to Cath Lab where therapeutic hypothermia will be started in conjunction with heart catheterization and/or percutaneous coronary intervention. 6. The physician will identify potential candidates for this protocol using the inclusion and exclusion criteria. a. Inclusion Criteria 1) Cardiac arrest with return of spontaneous circulation (ROSC) < 6 hours prior to induction of hypothermia 2) Unresponsive or not following commands following cardiac arrest 3) Age > 18 years but < 85 Page 1 of 12

Protocol: 4) Core temperature > 35 0 C following return of circulation 5) Witnessed arrest with a down time of less than 60 minutes 6) Stable hemodynamics with or without external support (i.e. pressors or Intraaortic Balloon Pump) b. Exclusion Criteria 1) Pregnancy 2) Existing DNR/DNI status 3) End stage terminal illness 4) End Stage Renal Disease 5) Sustained refractory ventricular dysrhythmias 6) Severe bradycardia without a temporary pacemaker 7) Active bleeding (GI or Intracerebral Hemorrhage ICH) 8) Traumatic full arrest 9) Severe neurologic dysfunction (i.e. dementia)or vegetative state prior to cardiac arrest 10) Platelet count < 50,000 11) Awake and responsive to verbal commands following cardiac arrest 12) Major surgery within 2 weeks 13) Sepsis 1. The Emergency Department will assess patients for appropriateness of treatment, and review inclusion and exclusion criteria. 2. Consult intensivist for admission to critical care. 3. Upon receipt of the physician order to initiate hypothermia protocol, assure the patient has the following supportive equipment: a. Endotracheal intubation and mechanical ventilator. b. Continuous ECG monitoring, pulse oximeter. c. Non-invasive BP monitoring. d. CVP and/or PA catheter to assess fluid volume/ hemodynamic status. Goal CVP 6-10 mm Hg or PCWP 8-12 mm Hg. Page 2 of 12

e. Arterial catheter for invasive BP monitoring if needed. f. Esophageal probe or Foley catheter with a temperature probe. g. Arctic Sun Cooling machine and appropriate size pads to ensure 40% of the body surface area is covered. Obese patients need the large pads. 4. Assess the patient s clinical status, prior to initiating hypothermia protocol. a. Obtain 12 Lead ECG. b. Obtain vital signs and initial labs. 1) Obtain continuous core temperature via esophageal probe or bladder thermistor. 2) Obtain HR, BP and MAP. 3) Assess rhythm with baseline EKG. 4) Assess respiratory rate and ventilator function. Because of peripheral vasoconstriction, pulse oximetry is often unreliable, and BGs will have to be done to assess oxygenation. 5) Assess baseline laboratory values as appropriate including: ABG, CBC, PT/PTT, INR, BMP, BNP, Mg, phosphorus, lactate, amylase, lipase, cardiac enzymes, and urine HCG (females 10-60 years old). c. Assess baseline level of consciousness and neurologic status. 1) Glasgow Coma Scale (GCS) 2) If possible obtain information on neurologic functioning from family prior to decision on hypothermia. Pittsburgh Outcome Categorization Scale or CPC-OPC Scale (See Appendix 1) Modified Rankin Scale (See Appendix 2) 3) Assess agitation using the Richmond Agitation and Sedation Scale or RASS (See Appendix 3) 4) Consider obtaining a stat CT scan of brain without contrast prior to cath lab procedures. d. Assess skin integrity prior to application of cooling pads. Page 3 of 12

5. Cooling Phase: Initiating hypothermia (preferably within 60 minutes of arrest) a. Initiate cooling as soon as possible after arrest. b. Infuse 30 ml/kg (maximum of 2 L) of ice cold (refrigerated - approx 4 0 C) saline solution over 30 minutes peripherally if no contraindication per physician s discretion. Do not infuse cold solution via subclavian or central line. Saline will be stored in Blood Bank. 1) Monitor Temperature every 15 minutes during infusion 2) Set up Arctic Sun and apply pads. DO NOT start Arctic Sun cooling until ice cold saline infusion is completed (external cooling interferes with Arctic Sun cooling algorithm) c. Obtain physician order for initiation of sedation and analgesia for cooling. Anticipate need for BP support. 1) Administer Morphine 2-4 mg IV per hour or Fentanyl 1-3 mcg/kg/hr IV per physician order. Morphine may decrease cardiac demand and fentanyl is a useful adjunct in shivering. 2) Start Propofol infusion at 10 mcg/kg/min and increase by 5 mcg/kg/min every 10 minutes to achieve a RASS of (-4). Maximum dose of 200 mcg/kg/min. Assess for metabolic acidosis > 50 mcg/kg/min. Refer to Critical Care Policy CC.E.31, Propofol (Diprivan) Infusion. 3) Monitor BP/MAP. The goal is > 80 and < 100 mm Hg. Use fluids to maintain euvolemia. Target CVP 6-10 mmg Hg or PCWP 8-12 mm Hg. Use vasopressors or vasoactive medication to increase MAP once euvolemic. If EF normal, neosynephrine or norepinephrine may be used to increase MAP. If EF is reduced, use dobutamine drip to reach MAP goal. If MAP falls despite dobutamine, add dopamine or epinephrine. Consider giving packed red blood cells to increase hemoglobin >10 and increase dobutamine. Page 4 of 12

d. Place continuous temperature monitoring probe (foley or esophageal) and attach to Arctic Sun cooling machine. e. Apply Arctic Sun pads to body and use the automatic mode on the Arctic Sun cooling machine. Connect pads to hose. 1) Set the desired target temperature to 33 0 C, Press Automatic, and begin cooling. 2) Follow the instructions on the machine for cooling. Assure system setup on Arctic Sun is set for hypothermia protocol. 3) Note time cooling is initiated on the flow sheet or patient s medical record. f. Obtain physician orders for the following medications for initial paralysis: 1) Ensure patient is adequately sedated prior to administration of paralytic. 2) Loading dose: Prior to induction of hypothermia, administer paralytic agent IV push, Tracurium (atracurium) 0.3-0.5 mg/kg IV bolus or Norcuron (vecuronium) 0.08 0.1 mg/kg, with a goal of 1-2 twitches with 4 shocks. 3) Maintenance dose: Tracurium (atracurium) continuous IV infusion at 4-12 mcg/kg/min or Norcuron (vecuronium) 0.8-1.7 mcg/kg/min. Retest TOF 1 hour after the start of continuous infusion and 1 hour after any NMBA dose adjustment, then q1h until the target TOF is achieved. Monitor TOF every 4 hours after the target TOF is achieved. Paralytic will also reduce the incidence of shivering as hypothermia is induced. g. Assess vital signs, ECG, SpO2, and presence of shivering every 15 minutes during induction of hypothermia, then every 30 minutes x2 hours, then every hour. 1) Document RASS every 30 minutes during induction and then every hour. 2) Monitor Arctic Sun water temperature and patient s core temperature every one hour. Page 5 of 12

h. Goal core temperature of 33-34 0 C should be achieved in 4-6 hours. Notify MD if unable to reach goal temperature within 6 hours. Younger patient respond to cooling with greater intensity than older patients do and may have exaggerated shivering and vaso-constrictive responses in greater proportion to the elderly. Obese patients will cool slower due to the insulating property of adipose tissue. i. Note TIME when patient achieved goal core temperature and document. Maintain goal temperature for 18 hours or a total of 24 hours since initiation of hypothermia. If patient demonstrates hemodynamic instability at 33-34 degrees Celsius, notify physician, stop cooling and initiate rewarming. j. Assess environment to facilitate cooling. 1) Discontinue the heater on the ventilator circuit. 2) Pull down window shades to block sunlight. 3) Discontinue heat lamps. 4) Turn off all unnecessary overhead lighting. 5) Set room thermostat to 68 O F. k. Obtain order for stress ulcer prophylaxis: Famotidine (Pepcid) 20 mg q 12 hours. l. Place knee high sequential compression device on legs or consider Enoxaparin (Lovenox) for DVT prophylaxis. m. Obtain cardiac echocardiogram. n. Monitor for arrhythmias, if unstable ventricular arrhythmias persist, contact physician for orders regarding appropriate anti-arrhythmic agent. Consider Amiodarone drip if no contraindications. 6. Assess for shivering refractory to vecuronium infusion and propofol (Diprivan). Utilize Shivering Assessment Scale (Appendix 4). a. Meperidine (Demerol) 25 mg IVP followed by a meperidine 25mg IVP every 30 minutes PRN shivering. Maximum dose is 500 mg/24 hours. Page 6 of 12

b. Vecuronium 0.1 mg/kg IVP every hour PRN shivering. 7. Ongoing assessment and monitoring a. Vital signs and temperature as described above. b. Continuous ECG monitoring. c. Assess for signs of shivering. If present, institute interventions as described above. d. Reassess skin at sites covered by the gel pads every 4 hours for cold related injuries. e. Assess urine output q 1 hour. f. Assess agitation using the RASS. g. Assess blood glucose q 1 hour. Initiate insulin intravenous order set as needed to keep blood glucose between 140-180 mg/dl. h. Obtain CBC, PT/PTT, CPK, MB, Troponin, ABG, lactate, and BMP at 6 hours, 12 hours, 18 hours, and 24 hours from initiation of cooling. i. Notify MD for: 1) Abnormal lab results 2) MAP < 60 mmhg 3) Inabilitiy to reach temp within 6 hours 4) Overshooting core temp (< 32 0 C) 5) ST elevation or depression 6) Urine output < 30 ml/hr. j. Monitor water temperature of the machine and patient s temperature every hour. 8. During initiation of hypothermia, the patient with normal renal function will increase urine output causing a potential loss of sodium, potassium, magnesium, phosphorus, and calcium. a. Hypokalemia and hypomagnesemia may exacerbate or cause ventricular arrhythmias. b. Elevated lactate levels may indicate metabolic acidosis. Page 7 of 12

c. Other complications of Hypothermia Effects of Hypothermia 30-35 O C Somatic: Shivering, peripheral vasoconstriction, increased muscle activity Metabolic: Increased fat metabolismlactate, metabolic acidosis Endocrine: insulin sensitivity, cortisol and adrenaline, insulin secretion = contribute to elevated blood glucose levels Cardiovascular: Tachycardia (35-36 O C) Bradycardia (<35 O C) CVP, cardiac output BP (vasoconstriction and afterload) Effects of Hypothermia 30-35 O C Mild arrhythmia (< 32 O C), risk of tachyarrhythmias (beginning with atrial fib -(28-30 O C) ECG changes: PR interval, widening QRS, QT interval, J waves Renal: Increased dieresis leading to electrolyte and volume loss. Intervention Fentanyl, Sedatives, Propofol, Meperidine, Neuromuscular Blockade Draw lactacte and chemistry panels during hypothermia and monitor for acidosis, assess propofol use, consider pan-culturing patient Monitor blood glucose hourly and maintain between 140-180 mg/dl Observe for adequate cardiac output and perfusion, monitor H & H Intervention Consider antiarrhythmic, maintain temp 33-34 O C Monitor ECG continuously Monitor and replace electrolytes. Monitor BP, CVP, replace fluid Page 8 of 12

GI: Impaired motility/ileus Liver dysfunction Mild pancreatitis Hematologic: platelet count, impaired leukocyte function, impaired macrophage function as needed. Monitor for ileus. Monitor liver enzymes. Monitor amylase and lipase. Monitor platelet count Monitor lactate and microbiology test result for infection and sepsis. 9. Use sliding scale to replace potassium if < 3.2 mmol/l a. KCL 40 meq IVPB (over 4 hours if peripheral IV or 2 hours if central line) for K < 3.2 mmol/l. b. KCL 80 meq IVPB (over 8 hours if peripheral IV or 4 hours if central line for K < 2.0 mmol/l and notify physician. 10. Use sliding scale to replace magnesium if < 2 mg/dl mmol/l a. Magnesium sulfate 2 Gm IVPB over 1 hour for magnesium < 2 mg/dl. 11. Rewarming Phase a. Rewarming is to begin: 1) 18 hours after target temperature is achieved or 24 hours after initiation of cooling 2) The hypothermia protocol has been discontinued by the physician 3) Platelet count decreases to < 50,000 4) Patient becomes hemodynamically unstable 5) Unstable arrhythmias persist b. Discontinue any electrolyte infusion 4 hours before rewarming. Notify MD if K < 3.0. c. Utilize the automatic mode on the Arctic Sun Cooling Machine and follow manufacturer s guidelines. Page 9 of 12

d. Set the desired temperature to 37 O C. Two nurses will be required to set the temperature and rate of rewarming on the machine to ensure appropriate settings. 1) Set the temperature to be increased to 0.5 O C every hour until 37 degrees Celsius is achieved. 2) Monitor ECG, vital signs, and SpO2 every 30 minutes during the rewarming process. Once target temperature is achieved, continue to monitor core temp hourly for 48 hours. 3) Monitor for signs and symptoms of hypovolemia and decrease in MAP/BP due to vasodilation. 4) Monitor for changes in electrolytes. Assess BMP, Mg, Phos, and Ca every 2 hours during rewarming. 5) If hyperkalemia develops during rewarming, decrease Arctic Sun set temperature by 0.5 O C and notify physician. e. Warm to normal body temperature no faster than 1 O C/hour. f. Assess for arrhythmias during rewarming phase. g. Once patient s core temperature reaches 36 O C, titrate down propofol off, and neuromuscular blocker per physician s order. h. Assess for rebound hyperthermia. i. Continue to utilize the Arctic Sun Cooling machine to maintain normothermia (37.0-37.5 O C) for 24 to 48 hours. Documentation: Document assessments and interventions in patient s medical record. References: Arrich, J. (2007). Clinical Application of Mild Therapeutic Hypothermia after Cardiac Arrest, Critical Care Medicine. 35 (4), 1041-1047. Badjatia, N., Strongilis, E., Gordon, E., Prescutti, M., Fernandez, L., Fernandez, A., et al. (2008), Metabolic Impact of Shivering during Therapeutic Temperature Modulation: The Bedside Shivering Assessment Scale. Stroke, 39, 3242-3247 Page 10 of 12

Bernard, S., Gray, T., Buist, M., Jones, B., Silvester, W., Gutteridge, G. et al. (2002). Treatment of Comatose Survivors of Out-of-Hospital Cardiac Arrest with Induced Hypothermia, New England Journal of Medicine, 346 (8), 557-563. Cincura, C., Pontes-Neto, O., Neville, I., Mendes, H., Menezes, D., Mariano, D., et al. (2009). Validation of the National Institutes of Health Stroke Scale, Modified Rankin Scale and Barthel Index in Brazil: The Role of Cultural Adaptation and Structured Interviewing, Cerebrovascular Diseases, 27 (2), 119-122 Keresztes, P., & Brick, K. (2006), Therapeutic Hypothermia after Cardiac Arrest, Dimensions of Critical Care Nursing. 25(2), 71-76. Holzer, A. & Mullner H. (2009), Hypothermia for Neuroprotection in Adults after Cardiopulmonary Resuscitation, Cochrane Collaboration, Wiley Publisher. Hypothermia after Cardiac Arrest (HACA) Study Group (2002), Mild Therapeutic Hypothermia to Improve the Neurologic Outcome after Cardiac Arrest, New England Journal of Medicine. 346 (8), 549-556. Nolan, J., Morley, P. Vanden Hoek, T, Hickey, R, Kloeck, W., Billi, K. et al. (2003). Therapeutic Hypothermia after Cardiac Arrest: An Advisory Statement by the Advanced Life Support Task Force of the International Liaison Committee on Resuscitation. Circulation, 108, 118-121. Oddo, M., Schaller, M., Feihl, F., Ribordy, V., & Liaudet, L. (2006), From Evidence to Clinical Practice: Effective Implementation of Therapeutic Hypothermia to Improve Patient Outcome after Cardiac Arrest, Critical Care Medicine. 34 (7), 1865-1873. Quinn, R., Dawson, J., Walters, M., & Lees, K. (2009), Reliability of the Modified Rankin Scale. Stroke, 3393-3395. Page 11 of 12

Submitted by: Alfie Ignacio, Marie Herbrandson *************** Initial Approvals and Major Revisions: Nursing Policy/Procedure Committee 2/01/10 Critical Care Committee 3/11/10 Cardiology Committee 3/01/10 Medicine Department 3/16/10 Emergency Department 4/15/10 Infection Control Pharmacy & Therapeutics Committee 5/11/10 Medical Executive Committee 5/11/10 Operations Committee 5/26/10 Initial Effective Date: 6/01/10 Revised Effective Date(s): Reviewed Date(s): Revised Date(s): Attachments: Appendix 1 - Glasgow-Pittsburgh Outcome Categorization Scale CPC-OPS Scale Appendix 2 - Modified Rankin Scale Appendix 3 - Richmond Agitation Sedation Scale (RASS) Appendix 4 - Bedside Shivering Assessment Scale (BSAS) Page 12 of 12

Appendix 1 Glasgow-Pittsburgh Outcome Categorization Scale CPC-OPS Scale Cerebral Performance Categories (CPC) Score Overall Performance Categories (OPC) 1 Good cerebral performance: Conscious, alert, and able to work and lead a normal life. Might have minor psychological or neurological deficits (mild dysphasia, noncapacitating hemiparesis, or minor cranial nerve abnormalities. 2 Moderate cerebral disability: Conscious. Sufficient cerebral function for part-time work in a sheltered environment or independent activities of daily life (dress, travel by public transportation, food preparation). Such patients may have hemiplegia, seizures, ataxia, dysarthria, dysphasia, or permanent memory or mental changes. 3 Severe cerebral disability: Conscious, patient dependent on others for daily support because of impaired brain function. Has at least limited cognition. It includes patients that are ambulatory but have severe memory disturbance or dementia precluding independent existence. 4 Coma/Vegetative State: Not conscious, unaware of surroundings, no cognition. No verbal and/or psychological interaction with environment. 5 Brain death: Certified brain dead or dead by traditional criteria Score (1-5): Reference: Jennett, B., & Bond, M. (1975). Assessment of outcome after severe brain damage. Lancet, 1 (7905): 480 484. Teasdale, G., & Jennett, B. (1974). Assessment of coma and impaired consciousness: a practical scale. Lancet, 2 (7872): 81 84.

Appendix 2 Modified Rankin Scale Score 0 No symptoms at all Description 1 No significant disability despite symptoms; able to carry out all usual duties and activities 2 Slight disability; unable to carry out all previous activities, but able to look after own affairs without assistance 3 Moderate disability; requiring some help, bet able to walk without assistance 4 Moderately severe disability; unable to walk without assistance and unable to attend to own bodily needs without assistance 5 Severe disability; bedridden, incontinent and requiring constant nursing care and attention 6 Dead Score (0-6): Reference: Cincura, C., Pontes-Neto, O., Neville, I., Mendes, H., Menezes, D., Mariano, D., et al. (2009). Validation of the National Institutes of Health Stroke Scale, Modified Rankin Scale and Barthel Index in Brazil: The Role of Cultural Adaptation and Structured Interviewing. Cerebrovascular Diseases, 27 (2), 119-122 Quinn, R., Dawson, J., Walters, M., & Lees, K. (2009). Reliability of the Modified Rankin Scale. Stroke, 3393-3395

Richmond Agitation Sedation Scale (RASS) * Appendix 3 Score Term Description 4 Combative Overtly combative, violent, immediate danger to staff 3 Very agitated Pulls or removes tube(s) or catheter(s); aggressive 2 Agitated Frequent non-purposeful movement, fights ventilator 1 Restless Anxious but movements not aggressive vigorous 0 Alert and calm -1 Drowsy Not fully alert, but has sustained awakening (eyeopening/eye contact) to voice (>10 seconds) -2 Light sedation Briefly awakens with eye contact to voice (<10 seconds) -3 Moderate sedation Movement or eye opening to voice (but no eye contact) -4 Deep sedation No response to voice, but movement or eye opening to physical stimulation -5 Unarousable No response to voice or physical stimulation Verbal Stimulation Physical Stimulation Procedure: 1. Observe patient a. Patient is alert, restless, or agitated. (score 0 to +4) 2. If not alert, state patient s name and say to open eyes and look at speaker. a. Patient awakens with sustained eye opening and eye contact. (score 1) b. Patient awakens with eye opening and eye contact, but not sustained. (score 2) c. Patient has any movement in response to voice but no eye contact. (score 3) 3. When no response to verbal stimulation, physically stimulate patient by shaking shoulder and/or rubbing sternum. a. Patient has any movement to physical stimulation. (score 4) b. Patient has no response to any stimulation. (score 5) Reference: Sessler CN, Gosnell M, Grap MJ, Brophy GT, O'Neal PV, Keane KA et al. The Richmond Agitation-Sedation Scale: validity and reliability in adult intensive care patients. Am J Respir Crit Care Med 2002; 166:1338-1344. Ely EW, Truman B, Shintani A, Thomason JWW, Wheeler AP, Gordon S et al. Monitoring sedation status over time in ICU patients: the reliability and validity of the Richmond Agitation Sedation Scale (RASS). JAMA 2003; 289:2983-2991.

Appendix 4 Bedside Shivering Assessment Scale (BSAS) Score Definition 0 None: no shivering noted on palpation of the masseter, neck, or chest wall 1 Mild: shivering localized to the neck and/or thorax only 2 Moderate: shivering involves gross movement of the upper extremities (in addition to neck and thorax 3 Severe: shivering involves gross movements of the trunk and upper and lower extremities Procedure: Observe the patient for a period of 2 minutes, during which time visually inspect and palpate the neck, thorax, arms and legs. Reference: Badjatia, N., Strongilis, E., Gordon, E., Prescutti, M., Fernandez, L., Fernandez, A., et al. (2008). Metabolic impact of shivering during therapeutic temperature modulation: The Bedside Shivering Assessment Scale. Stroke, 39; 3242-3247