Interpreting AV (Heart) Blocks: Breaking Down the Mystery
|
|
|
- Darren Berry
- 10 years ago
- Views:
Transcription
1 Interpreting AV (Heart) Blocks: Breaking Down the Mystery 2 Contact Hours Copyright 2012 by RN.com. All Rights Reserved. Reproduction and distribution of these materials is prohibited without the express written authorization of RN.com. Course Expires: 12/13/2015 First Published: 9/13/2012
2 Acknowledgments RN.com acknowledges the valuable contributions of...kim Maryniak, RNC-NIC, BN, MSN. Kim has over 23 years staff nurse and charge nurse experience with medical/surgical, psychiatry, pediatrics, and neonatal intensive care. She has been an educator, instructor, and nursing director. Her instructor experience includes med/surg nursing, mental health, and physical assessment. Kim graduated with a nursing diploma from Foothills Hospital School of Nursing in Calgary, Alberta in She achieved her Bachelor in Nursing through Athabasca University, Alberta in 2000, and her Master of Science in Nursing through University of Phoenix in Kim is certified in Neonatal Intensive Care Nursing and is currently pursuing her PhD in Nursing. She is active in the National Association of Neonatal Nurses and American Nurses Association. Kim s previous roles in professional development included nursing peer review and advancement, teaching, and use of simulation. Her current role as clinical director provides oversight of travel and per diem nurses, including education, quality, and process improvement. Disclaimer RN.com strives to keep its content fair and unbiased. The author(s), planning committee, and reviewers have no conflicts of interest in relation to this course. There is no commercial support being used for this course. Participants are advised that the accredited status of RN.com does not imply endorsement by the provider or ANCC of any commercial products mentioned in this course. There is "off label" usage of medications discussed in this course. You may find that both generic and trade names are used in courses produced by RN.com. The use of trade names does not indicate any preference of one trade named agent or company over another. Trade names are provided to enhance recognition of agents described in the course. Note: All dosages given are for adults unless otherwise stated. The information on medications contained in this course is not meant to be prescriptive or all-encompassing. You are encouraged to consult with physicians and pharmacists about all medication issues for your patients. Purpose The purpose of this course is to discuss the essentials of atrioventricular block rhythms to identify the differences between the degrees. Some review of basic ECG interpretation will be included, as well as causes of the blocks. AV Blocks vs. Heart Blocks The terms AV Block and Heart Block are synonymous. AV Block is the most current and most correct term, but you may still hear Heart Block used in clinical practice.
3 Learning Objectives After successful completion of this course, you will be able to: 1. Describe the basic electrophysiology of the heart. 2. Identify basic components of an ECG strip. 3. Describe the causes of atrioventricular heart blocks. 4. Distinguish between the degrees of heart blocks by identifying rhythm strips. Introduction Telemetry interpretation is an important skill for nurses in many patient care areas. Lethal arrhythmias may be readily identified, but other rhythms may not be as easily recognized. Atrioventricular (AV) blocks may not be commonly encountered in patient care. Breaking down the components of the different blocks is valuable to interpret the various AV blocks. This course focuses on AV block interpretation. Standard lead selection, placement, tracings, treatment, and lethal arrhythmias are not covered in this course. If you need to review these topics, please refer to the Telemetry Interpretation and Lethal Arrhythmias: Advanced Rhythm Interpretation courses on RN.com. Review: Electrophysiology of the Heart Two distinct components must occur for the heart to be able to contract and pump blood. These components are: (1) An electrical impulse and (2) A mechanical response to the impulse. 1. The electrical impulse tells the heart to beat through automaticity. Automaticity means that these specialized cells within the heart can discharge an electrical current without an external pacemaker or stimulus from the brain via the spinal cord. The electrical (conductive) cells of the heart initiate electrical activity. 2. The mechanical beating or contraction of the heart occurs after the electrical stimulation. When the mechanical contraction occurs the person will have both a heart rate and a blood pressure. Specific mechanical (contracting) cells respond to the stimulus and of the electrical cells and contract to pump blood.
4 Review: Depolarization & Repolarization In a cardiac cell, two primary chemicals provide the electrical charges: sodium (Na+) and potassium (K+). In the resting cell, most of the potassium is on the inside, while most of the sodium is on the outside. This results in a negatively charged cell at rest (the interior of the cardiac cell is negative or polarized at rest). When depolarized, the interior cell becomes positively charged and the cardiac cell will contract. Since the polarized or resting cell has the negative charge on the inside at rest, depolarization occurs when potassium and sodium move across the cell membrane and the inside of the cell becomes positively charged. As depolarization occurs, the change in membrane voltage triggers contraction of the cell. Depolarization moves a wave through the myocardium. As the wave of depolarization stimulates the heart s cells, they become positive and begin to contract. This cell-to-cell conduction of depolarization through the myocardium is carried by the fast moving sodium ions. Repolarization is the return of electrical charges to their original state. This process must happen before the cells can be ready to conduct again. Knowledge Check 1 Repolarization is defined as: A. The positive charge of cells. B. The system of conduction. C. The return of electrical charges to their original state. (correct) Review: The Cardiac Conduction System The specialized electrical cells in the heart are arranged in a system of pathways called the conduction system. These specialized electrical cells and structures guide the wave of myocardial depolarization. The conduction system consists of the sinoatrial node (SA node), atrioventricular node (AV node), bundle of His (also called the AV junction), right and left bundle branches, and Purkinje fibers.
5 Review: The Sinoatrial (SA) Node The sinoatrial node (also called the SA node or sinus node) is a group of specialized cells located in the posterior wall of the right atrium. The SA node normally depolarizes or paces more rapidly than any other part of the conduction system. It sets off impulses that trigger atrial depolarization and contraction. The SA node normally fires at a rate of beats per minute. After the SA node fires, a wave of cardiac cells begin to depolarize. Depolarization occurs throughout both the right and left atria (similar to the ripple effect when a rock is thrown into a pond). This impulse travels through the atria by way of inter-nodal pathways down to the next structure, which is called the AV node. Review: The Atrioventricular (AV) Node and AV Junction The next area of conductive tissue along the conduction pathway is at the site of the atrioventricular (AV) node. This node is a cluster of specialized cells located in the lower portion of the right atrium above the base of the tricuspid valve. The AV node itself possesses no pacemaker cells. The AV node has two functions. The first function is to DELAY the electrical impulse in order to allow the atria time to contract and complete the filling of the ventricles. The second function is to receive an electrical impulse and conduct it down to the ventricles via the AV junction and bundle of His. Review: The Bundle of His After passing through the AV node, the electrical impulse enters the bundle of His (also referred to as the common bundle). The bundle of His is located in the upper portion of the intraventricular septum and connects the AV node with the two bundle branches. If the SA node becomes diseased or fails to function properly, the bundle of His has pacemaker cells that are capable of discharging at an intrinsic rate of beats per minute. The AV node and the bundle of His are referred to collectively as the AV junction. The bundle of His conducts the electrical impulse down to the right and left bundle branches. The bundle branches further divide into Purkinje fibers. Review: The Purkinje Fibers At the terminal ends of the bundle branches, smaller fibers distribute the electrical impulses to the muscle cells, which stimulate contraction. This web of fibers is called the Purkinje fibers. The Purkinje fibers penetrate 1/4 to 1/3 of the way into the ventricular muscle mass and then become continuous with the cardiac muscle fibers. The electrical impulse spreads rapidly through the right and left bundle branches and Purkinje fibers to reach the ventricular muscle, causing ventricular contraction, or systole. The Purkinje fibers within the ventricles also have intrinsic pacemaker ability. This third and final pacemaker site of the myocardium can only pace at a rate of beats per minute. Notice that the further you travel away from the SA node, the slower the backup pacemakers
6 become. If you only have a heart rate of 30 (from the ventricular back-up pacemaker), blood pressure will likely be low and the patients will likely be quite symptomatic. Knowledge Check 2 The conduction system of the heart includes the SA node, the AV node and: A. Bundle of His B. Right and left bundle branches C. Purkinje fibers D. All of the above (correct) ECG: Review of the Basics Before jumping in to interpreting AV blocks, a quick review of the basics for ECG strips is needed. The ECG is a recording of the electrical impulses produced by the heart. The ECG strip is standardized, with time measured in seconds along the horizontal axis. Each small square is 1 mm in length and represents 0.04 seconds. Each larger square is 5 mm in length and therefore represents 0.20 seconds. The body acts as a giant conductor of electrical currents. The tracing recorded from the electrical activity of the heart forms a series of waves and complexes that have been arbitrarily labeled (in alphabetical order) the P, Q, R, S, and T waves. The six second method can be used with either regular or irregular rhythms and provides a rough estimate (but not precise) of heart rate. Print a 6 second strip, count the number of R waves in a six second strip and multiply by 10. For example, if there are seven (7) R waves in a six second strip, the heart rate is approximately 70 or (7x10=70).
7 ECG: The P Wave Electrical impulses originating from the SA node are represented on the ECG with a waveform called a P wave. The P wave is generated after the SA node fires and depolarizes the right and left atria. The beginning of the P wave is recognized as the first upward deflection from the baseline. It resembles a small upward hill or bump and once completed, returns to the ECG baseline. Normal ECG Waveforms and Intervals ECG: The PR Interval When the impulse leaves the atria and travels to the AV node it encounters a slight delay. The tissues of the node do not conduct impulses as fast as the other cardiac electrical tissues. This means that the wave of depolarization will take a longer time to get through the AV node. On the ECG this is represented by a short period of electrical inactivity called the PR interval. The PR interval extends from the beginning of the P wave (the beginning of atrial depolarization) to the onset of the QRS complex (the beginning of ventricular depolarization). The PR interval (or time travel from SA to AV nodes) is between 0.12 to 0.20 seconds. It should not exceed 0.20 seconds as measured on ECG graph paper where each small square represents 0.04 seconds. It is actually only measured from the beginning P to the beginning of the Q wave (think of it as a P to Q measurement despite the fact that it is called a PR interval). Changes in conduction through the AV node are the most common cause of changes in the PR interval. The P to R interval is important in identification of heart blocks. ECG: The QRS Complex The ventricular depolarization is shown on the ECG by a large complex of three waves: the Q, the R, and the S waves. Together, these three waves are called the QRS complex. The QRS voltage or amplitude is much higher than the height of the P wave. This is because ventricular depolarization involves a greater muscle mass and creates a larger complex. Following the P wave, the Q wave is the first negative or downward deflection. The R wave is the first positive or upward deflection following the P wave. The negative wave following the R wave is known as the S wave. Each QRS complex can look a bit different. In fact, some QRS complexes are lacking a Q wave or
8 others may lack the S wave. Regardless of the appearance, they are always generically called the QRS and still indicate depolarization of the ventricles. The upper limit of normal duration of the QRS complex is less than 0.12 seconds or three small boxes. Place one leg of your caliper on the beginning of the Q wave and place the other leg of the caliper on the S wave where it meets the ST segment. ECG: ST Segment and T Wave The ST segment begins at the end of the S complex and ends with the onset of the T wave. The ST segment represents the early part of repolarization of the ventricles. The ST segment normally sits on the baseline or isoelectric line. It is also normal if the ST segment is slightly elevated or below the isoelectric line (no greater than one millimeter in either direction). Ventricular repolarization is represented on the ECG by a T wave. The beginning of the T wave is identified at the point where the slope of the ST segment appears to become abruptly or gradually steeper. The T wave ends when it returns to the isoelectric baseline. ECG: Steps to Rhythm Interpretation 1. Rate: Calculate the heart rate (HR) or note the HR from the monitor. 2. Regularity: Measure the regularity or rhythm of the R waves. 3. P-wave Examination: Is there one P wave before each QRS? (there should be) 4. P to R Interval: Measure the P to R interval - Is it within normal limits? It is consistent? 5. QRS Width: Measure the duration of the QRS complex. Knowledge Check 3 A normal PR interval is: A seconds B. Less than 0.12 seconds C. Between 0.12 and 0.20 seconds (correct) Atrioventricular Blocks Atrioventricular (AV) blocks arise when atrial depolarizations do not reach the ventricles or when there is a delay in atrial depolarization conductions. There are three degrees of AV block: First degree Second degree (in two forms) Third degree
9 When learning and attempting to memorize information, the use of acronyms, mnemonics, or analogies are useful. For the discussion of AV blocks, the analogy of a bus driver will be used. The bus driver is the P wave, the PR interval is how long it takes to get to the bus stop, and the QRS complex are the passengers that are picked up on the bus. The use of visual depictions of the bus along the bus route will help identify AV blocks on ECG strips. Symptoms of AV Blocks Signs and symptoms depend on the type of AV block that is occurring. First-degree AV block rarely causes symptoms. Symptoms of second- and third-degree AV block include: Fainting Dizziness Fatigue Shortness of breath Chest pain First Degree AV Block Unlike its name (which can be confusing), first-degree AV block is not an actual block, but rather a delay in conduction. Therefore, first-degree AV block is simply a delay in passage of the impulse from atria to ventricles. This conduction delay usually occurs at the level of the AV node. Remember that in normal sinus rhythm, the time it takes the SA node to fire, depolarize the atria and transmit to the AV node is <.20 seconds. In first degree AV block the patient has a PR interval of >.20 seconds. Rate: Not affected; varies depending on the underlying rhythm Regularity: Regular P-Waves: Upright and normal. One P precedes every QRS PR Interval: Prolonged (>0.2 seconds) and is constant QRS Duration: Not affected ( 0.12 seconds) Causes of First Degree AV Block Drug therapy (digoxin, beta-adrenergic blockers or calcium channel blockers, or antiarrhythmic drugs such as amiodarone) Post-MI Chronic degenerative disease of the atrial conduction system (seen with aging) Hypo- or hyperkalemia Increased vagal tone
10 The Bus Driver in First Degree AV Block For this scenario of first-degree AV blocks, the bus driver is Frank. Frank always arrives to pick up his passengers (the QRS complex), but he is regularly late to arrive at the bus stop (PR interval). Although Frank keeps his customers waiting, at least he s consistent about it. Sinus Rhythm with First Degree AV Block Rate: approximately 80 Regularity: Atrial rhythm regular, ventricular rhythm regular P wave: each P wave is followed by a QRS complex PR interval: > 0.2 QRS: Normal, < 0.12
11 Treatment: First Degree AV Block A priority of the nurse is to observe for lengthening PR intervals or development of more serious heart blocks. Potential Treatments: Treatment for first-degree heart block is usually unnecessary as it is typically asymptomatic. Treatment typically aims to correct the underlying cause. If it is caused from drug therapy, then adjust or remove the medication. If it is a potassium imbalance, then it needs to be corrected. Consult with physician if PR interval is lengthening. Discuss holding medications which slow AV node conduction, such as beta-blockers or calcium channel blockers Second Degree AV Blocks There are two categories of second-degree heart block. One is called Wenckebach (Type I) and the other is called Type II. In both types, the impulse originates in the sinus node, but is conducted through the AV node in an intermittent fashion. Simply stated, not every P wave will be followed by QRS complex. In second-degree heart blocks, some impulses are conducted and others are not. The cause of the non-conducted P waves is related to intermittent AV nodal block. The difference between the two-second degree blocks is related to the pattern in which the P waves are blocked. Second Degree AV Block Type I (Wenckebach) Second Degree AV Block Type II Two Types of Second Degree AV Blocks Second Degree AV Block Type I - Wenckebach Second-degree AV block-type I is unique in that it has three different names, and all three are used interchangeably: Second Degree AV Block-Type I, Mobitz I or Wenckebach. Do not let this confuse you; all three names mean the SAME rhythm. We will refer to Second Degree AV Block Type I as Wenckebach for the remainder of this course. Wenckebach is characterized by a progressive prolongation of the PR interval (so the key to diagnosing this rhythm is by careful examination of each PR interval). The SA node is healthy and fires on time, thus the P to P intervals are regular. Impulses traveling through the AV node take longer and longer to fully conduct until one impulse is completely blocked. The SA node continues to fire right on time (regular P to P intervals) and the cycle of prolongation of PR intervals continues as the pattern is repeated
12 Second Degree AV Block Type I - Wenckebach The repetition of this pattern results in group beating, (e.g. three conducted sinus beats with progressively lengthening PR intervals and a fourth sinus beat that is NOT followed by a QRS). Beats that are successfully conducted have a normal QRS width. Because QRS complexes are periodically dropped, the ventricular rhythm is irregular. This block almost always occurs at the level of the AV node (rarely at the bundle of His or bundle branch level), is typically a transient rhythm, and prognosis is good. Rate: Depends on the underlying atrial rate. Atrial regular; Ventricular rate is slightly slower. Typically between bpm Regularity: Regularly irregular; Atrial regular. Ventricular irregular P-Waves: More P-waves than QRS complexes, associated with each conducted QRS complex. PR Interval: Progressively lengthens until a QRS complex is dropped. After the blocked beat, the cycle starts again QRS Duration: Not affected ( 0.12 seconds) Causes of Second Degree AV Block Type I - Wenckebach Drug therapy (digoxin, beta-adrenergic blockers, calcium channel blockers, amiodarone) CAD New MI seen more commonly with acute inferior wall infarctions Rheumatic fever Increased vagal tone Hyperkalemia Myocarditis
13 The Bus Driver in Second Degree AV Block Type I - Wenckebach For this scenario of second-degree AV Block Wenckebach, the bus driver is Wendy. Wendy is late and forgetful. She arrives late to pick up passengers (QRS complex) at one stop, and is even later at subsequent stops (PR interval). She eventually forgets to pick up her passengers (a dropped QRS complex after a P wave), but then tries to get back on schedule. Wendy is late, later, later still, and then forgets her passengers altogether. Wendy s customer service skills are lacking, and she probably has several complaints against her. Sinus Rhythm with 2nd Degree AV Block - Wenckebach (Type I, Mobitz I) Rate: approximately irregular Regularity: Atrial rhythm regular, ventricular rhythm irregular P wave: each P wave is NOT followed by a QRS complex PR interval: increasing with each beat and then a P wave occurs without a QRS QRS: Normal, < 0.12
14 Treatment: Second Degree AV Block Type I - Wenckebach As a nursing priority, assess the patient for possible causes and monitor blood pressure, pulse, and other vital signs. Potential Treatments: Treatment is not typically required. Asymptomatic: Observation and monitoring only. If PR interval is lengthening, consult with physician for holding drugs that can slow AV node conduction. Symptomatic: On a rare occasion, atropine and/or temporary pacing may be considered. Knowledge Check 4 Characteristics of a second degree Type I Mobitz, or Wenckebach include: A. Widened QRS complex B. Progressive lengthening of the PR interval, with a dropped QRS complex (correct) C. A consistent delayed PR interval Second Degree AV Block Type II Second degree AV block Type II is also referred to as Mobitz II. This form of conduction delay occurs below the level of the AV node, either at the bundle of His (uncommon) or the bundle branches (common). A hallmark of this type of second-degree AV block is that there is a pattern of conducted P waves (with a constant PR interval), followed by one or more non- conducted P waves. The PR interval does not lengthen before a dropped beat. Remember that the P waves that are successful in conducting through have a constant PR interval. Since the SA node is firing in a regular pattern, the P to P intervals again march through in a regular pattern (P-P is regular). Since not all P waves are conducted into the ventricles, the R to R intervals will be irregular and the ventricular response (HR) may be in the bradycardia range. When the block occurs at the bundle of His, the QRS may be narrow since ventricular conduction is not disturbed in beats that are not blocked. If the blockage occurs at the level of the bundle branches, conduction through the ventricles will be slower therefore creating a wider QRS complex (>0.12 seconds). Mobitz II is associated with a poorer prognosis, and complete heart block may develop. Causes are usually associated with an acute myocardial infarction, severe coronary artery disease or other types of organic lesions in the conduction pathway. The patient s response to the dysrhythmia is usually related to the ventricular rate. Rate: Depends on the underlying atrial rate and the ratio of conducted complexes. Atrial regular; ventricular rate is typically ¼ to ½ the atrial rate (depending on the amount of blockage in conduction) Regularity: Regular, regularly irregular, or irregular (based on the ratio of conducted complexes). Atrial regular (P-P is regular). Ventricular irregular P-Waves: Upright and normal. Some P waves are not followed by a QRS (more Ps than QRSs). PR Interval: The PR interval for conducted beats will be constant across the strip QRS Duration: Not affected; >0.12 seconds for conducted beats
15 Causes of Second Degree AV Block Type II (Mobitz II) Age related degenerative changes in the conduction system New MI- seen more commonly with acute anterior wall infarctions Post cardiac surgery or complication arising with cardiac catheterization Note: not typically a result of increased vagal tone or drug effects The Bus Driver in Second Degree AV Block Type II (Mobitz II) For this scenario of second-degree Mobitz II AV blocks, the bus driver is Moe. Moe is never late (constant PR interval), but he makes more stops than is needed. He arrives on time to pick up passengers (QRS complex), but then makes stops where there are no passengers to board (a P wave without a QRS complex). Although Moe may be high in his efforts, he is not very efficient at his job. Second Degree AV Block Type II (Mobitz II) Rate: Ventricular approx 30; atrial approx 110 Regularity: Atrial rhythm regular, ventricular rhythm regular P wave: each P wave is NOT followed by a QRS PR interval: Consistent when followed by a QRS; < 0.2 QRS: Normal, < 0.12
16 Treatment: Second Degree AV Block Type II (Mobitz II) As a nursing priority, assess the patient for possible causes and monitor blood pressure, pulse, and other vital signs. Note: Mobitz II has the potential to suddenly progress to complete AV block (third degree AV block) or ventricular standstill; have a temporary pacemaker nearby! Potential Treatments: Asymptomatic: Observation and monitoring only. Hold drugs that can slow AV node conduction. Notify physician. Obtain supplies for pacing should this become necessary. Symptomatic: If symptomatic bradycardia is present, temporary pacing is initiated. Administer a dopamine infusion if patient is hypotensive. Note: Atropine must be used with great caution (if at all) with this rhythm. Atropine will increase the sinus note discharge, but does not improve conduction through the AV node, (the location of this block is lower in the conduction system). Acceleration of the atrial rate may result in a paradoxical slowing of the ventricular rate, thereby decreasing the cardiac output. Third Degree AV Block Third-degree AV block is also called Complete Heart Block. This type of dysrhythmia indicates complete absence of conduction between atria and ventricles (the atria and the ventricles are not communicating with one another). The atrial rate is always equal to or faster than the ventricular rate in complete heart block. The block may occur at the level of the AV node, the bundle of His, or in the bundle branches. Remember that the rhythm strip reflects two separate processes that are taking place. The SA node continues to control the atria and typically fires at a rate of bpm. Since the atria and the ventricles are not communicating, one of the two remaining back-up intrinsic pacemakers will take over. Either the junction will pace the ventricles (rate bpm) or the back-up ventricular pacer will discharge (rate bpm). On the ECG, you will see normal P waves marching regularly across the strip. The P-P intervals are regular. You will also see QRS complexes at regular intervals. The unique feature is that the P waves and the QRS complexes will not be talking to each other. There is no relationship between the P and the QRS waveforms. The PR interval will be totally inconsistent and you may even see P waves superimposed in the middle of QRS complexes. There will be more P waves than QRS complexes (because the intrinsic rate of the sinus node is faster than either the junctional or ventricular rates). Rate: Atrial rate will be independent of ventricular rate. Atrial rate is normal. Ventricular rate is slower bpm if back-up pacer is from the junction or bpm if back-up pacer is from the ventricles. Regularity: Atrial rate will be regular, ventricular rate will be regular. P-P is regular; R-R is regular (but the two are independent functions) P-Waves: Upright and normal. Present and disassociated from the ventricular activity PR Interval: Non-existent; No relationship between the P and the QRS waves. QRS Duration: 0.12 seconds if controlled by the junction; >0.12 seconds if paced by the ventricle.
17 Causes of Third Degree AV Block Drug therapy (digoxin, beta-adrenergic blockers, calcium channel blockers, amiodarone) New MI seen more commonly with acute inferior wall infarctions Increased vagal tone Hyperkalemia Myocarditis or rheumatic heart fever Post cardiac surgery or complication arising with cardiac catheterization The Bus Driver in Third Degree AV Block For this scenario of third-degree AV block, the bus driver is Thor. Thor tries very hard, but always misses his passengers. He does not have a consistent schedule (no PR interval), and so his passengers (QRS complex) end up trying to find their own ride. Thor probably won t have his job for much longer.
18 Third Degree AV Block Rate: Ventricular- approx. 30; atrial approx. 60 Regularity: Atrial rhythm regular, ventricular rhythm regular P wave: normal, disassociated from the QRS waves PR interval: irregular QRS: Normal, < 0.12 Treatment: Third Degree As a nursing priority, assess the patient for possible causes and monitor blood pressure, pulse, and other vital signs. Assess for syncope, palpitations, or shortness of breath. Hypotension may occur due to a low ventricular rate. For patient safety, lie your patient down to prevent syncope and potential falls. Potential Treatments: Asymptomatic: Notify physician. Observation and monitoring only. Hold drugs that can slow AV node conduction. Anticipate and obtain supplies for pacing should this become necessary. Symptomatic: Notify physician. If symptomatic bradycardia is present, administer atropine and prepare for temporary pacing. Atropine may be effective if the QRS is narrow (AV node level of block), but it has little or no effect on wide QRS (bundle-branch level) third-degree block rhythms. Administer a dopamine infusion if patient is hypotensive. A Note on Pacemakers Second degree AV Block Type II (Mobitz II) and Third-degree AV Block usually require temporary and/or permanent pacemakers. A second degree Mobitz II with a wide QRS complex indicate diffuse conduction system disease and is an indication for pacing even in the absence of symptoms. Mobitz II with a wide QRS may degenerate into third-degree AV block, which is another reason to consider permanent pacing. Knowledge Check 5 The main characteristic of a third degree AV block is: A. A regular P-P interval B. Prolonged PR interval with some P waves without QRS complexes C. No correlation between the P waves and the QRS complexes (correct)
19 Practice #1 Rate: Regularity: P-Waves: PR Interval: QRS Duration: What type of block is this? Practice #1 (answer) Rate: 30 bpm Regularity: P-P is regular; R-R is regular (but the two are independent functions) P-Waves: Upright and normal. Present and disassociated from the ventricular activity PR Interval: Non-existent; No relationship between the P and the QRS waves. QRS Duration: 0.12 seconds Answer: This is a third degree AV block
20 Practice #2 Note: This is a 3 second strip, rather than a standard 6 second strip Rate: Regularity: P-Waves: PR Interval: QRS Duration: What type of block is this? Practice #2 (answer) Note: This is a 3 second strip, rather than a standard 6 second strip Rate: 60 bpm Regularity: Regularly irregular; Atrial regular. Ventricular irregular P-Waves: More P-waves than QRS complexes, associated with each conducted QRS complex PR Interval: Progressively lengthening until a QRS complex is dropped. QRS Duration: 0.12 seconds Answer: This is a second degree AV block Type I Wenckebach
21 Practice #3 Rate: Regularity: P-Waves: PR Interval: QRS Duration: What type of block is this? Practice #3 (answer) Rate: 60 bpm Regularity: Atrial regular (P-P is regular). Ventricular irregular P-Waves: Upright and normal. Some P waves are not followed by a QRS (more Ps than QRSs) PR Interval: The PR interval for conducted beats will be constant across the strip QRS Duration: >0.12 seconds for conducted beats Answer: This is a second degree AV block Type II Mobitz II
22 Practice #4 Rate: Regularity: P-Waves: PR Interval: QRS Duration: What type of block is this? Practice #4 (answer) Rate: Variable; Regularity: Irregular P-Waves: Upright and normal. Some P waves are not followed by a QRS PR Interval: Variable; increasing with each beat and then a P wave occurs with no QRS following QRS Duration: < 0.12 seconds Answer: This is Second Degree AV Block; Mobitz I (Wenckebach)
23 Practice #5 Rate: Regularity: P-Waves: PR Interval: QRS Duration: What type of block is this? Practice #5 (answer) Rate: 30 bpm Regularity: Regular P-Waves: Upright and normal. One P precedes every QRS PR Interval: Prolonged (>0.2 seconds) and is constant QRS Duration: 0.12 seconds Answer: This is a bradycardia, with first degree AV block.
24 Practice #6 Rate: Regularity: P-Waves: PR Interval: QRS Duration: What type of block is this? Practice #6 (answer) Rate: less than 20 Regularity: P-P is regular, R-R is regular (but the two are independent functions) P-Waves: Upright and normal. Present and disassociated from the ventricular activity PR Interval: Non-existent; No relationship between the P and the QRS waves QRS Duration: < 0.12 seconds Answer: This is third degree AV block
25 Practice #7 Rate: Regularity: P-Waves: PR Interval: QRS Duration: What type of block is this? Practice #7 (answer) Rate: < 30 bpm Regularity: Atrial regular, ventricular regular P-Waves: Upright and normal. Some P waves are not followed by a QRS PR Interval: The PR interval for the conducted beats is consistent across the strip QRS Duration: < 0.12 seconds Answer: This is second degree AV block Type II Mobitz II
26 Practice #8 Rate: Regularity: P-Waves: PR Interval: QRS Duration: What type of block is this? Practice #8 (answer) Rate: Variable, from bpm Regularity: Irregular P-Waves: Upright and normal. Some P waves are not followed by a QRS PR Interval: Variable; increasing with each beat and then a P wave occurs with no QRS following QRS Duration: < 0.12 seconds Answer: This is Second Degree AV Block; Mobitz I (Wenckebach) Review of AV Blocks First-Degree AV Block: PR interval greater than 0.20 Second-Degree AV Block Type I, Mobitz I, Wenckebach: Progressively lengthening PR interval until one P wave is not followed by QRS Second-Degree AV Block Type II, Mobitz II: More P waves that QRS complexes; each QRS has a P wave preceding it; PR interval is consistent Third-Degree AV Block: P-P interval is regular; R-R interval is regular (but the two are independent functions). There is no relationship between P waves and QRS complexes, therefore there is no PR interval
27 Decision Tree
28 Conclusion AV blocks are not necessarily encountered on a daily basis with patient care, and can be a common area of difficulty in identifying the strips. Breaking down the components of the strips and relating them to analogies can be helpful to determine the differences between the degrees of blocks. Repeatedly viewing, reviewing, and interpreting these strips will assist in solidifying the knowledge of AV blocks. Resources Click here to view a short voice-over PowerPoint presentation on identification of 2nd and 3rd degree AV blocks. (URL to presentation: ) All materials copyrighted by RN.com Additional Resources University of Utah-School of Medicine. Alan E. Lindsay ECG Learning Center ECG Library Jenkins, J & Gerrend, S. (2002)
29 References Jones, S.A. (2008). ECG success: Exercises in ECG interpretation. Philadelphia: F.A. Davis. Marzlin, K. M. & Webner, C. L. (2006). Cardiovascular nursing: A comprehensive overview (1st ed.). Brockton, MA: Western Schools. Suraciwz, B., Childers, R., Deal, B. J. & Gettes, L. (2009). AHA/ACCF/HRS recommendations for the standardization and interpretation of the electrocardiogram. Circulation, 119, e235-e240.
ACLS Chapter 3 Rhythm Review Instructor Lesson Plan to Accompany ACLS Study Guide 3e
ACLS Chapter 3 Rhythm Review Lesson Plan Required reading before this lesson: ACLS Study Guide 3e Textbook Chapter 3 Materials needed: Multimedia projector, computer, ACLS Chapter 3 Recommended minimum
INTRODUCTORY GUIDE TO IDENTIFYING ECG IRREGULARITIES
INTRODUCTORY GUIDE TO IDENTIFYING ECG IRREGULARITIES NOTICE: This is an introductory guide for a user to understand basic ECG tracings and parameters. The guide will allow user to identify some of the
By the end of this continuing education module the clinician will be able to:
EKG Interpretation WWW.RN.ORG Reviewed March, 2015, Expires April, 2017 Provider Information and Specifics available on our Website Unauthorized Distribution Prohibited 2015 RN.ORG, S.A., RN.ORG, LLC Developed
Equine Cardiovascular Disease
Equine Cardiovascular Disease 3 rd most common cause of poor performance in athletic horses (after musculoskeletal and respiratory) Cardiac abnormalities are rare Clinical Signs: Poor performance/exercise
MULTIPLE CHOICE. Choose the one alternative that best completes the statement or answers the question.
Exam Name MULTIPLE CHOICE. Choose the one alternative that best completes the statement or answers the question. 1) What term is used to refer to the process of electrical discharge and the flow of electrical
Electrocardiography I Laboratory
Introduction The body relies on the heart to circulate blood throughout the body. The heart is responsible for pumping oxygenated blood from the lungs out to the body through the arteries and also circulating
VCA Veterinary Specialty Center of Seattle
An electrocardiogram (ECG) is a graph of the heart`s electrical current, which allows evaluation of heart rate, rhythm and conduction. Identification of conduction problems within the heart begins with
Introduction to Electrocardiography. The Genesis and Conduction of Cardiac Rhythm
Introduction to Electrocardiography Munther K. Homoud, M.D. Tufts-New England Medical Center Spring 2008 The Genesis and Conduction of Cardiac Rhythm Automaticity is the cardiac cell s ability to spontaneously
BASIC CARDIAC ARRHYTHMIAS Revised 10/2001
BASIC CARDIAC ARRHYTHMIAS Revised 10/2001 A Basic Arrhythmia course is a recommended prerequisite for ACLS. A test will be given that will require you to recognize cardiac arrest rhythms and the most common
School of Health Sciences
School of Health Sciences Cardiology Teaching Package A Beginners Guide to Normal Heart Function, Sinus Rhythm & Common Cardiac Arrhythmias Welcome This document extends subjects covered in the Cardiology
NEONATAL & PEDIATRIC ECG BASICS RHYTHM INTERPRETATION
NEONATAL & PEDIATRIC ECG BASICS & RHYTHM INTERPRETATION VIKAS KOHLI MD FAAP FACC SENIOR CONSULATANT PEDIATRIC CARDIOLOGY APOLLO HOSPITAL MOB: 9891362233 ECG FAX LINE: 011-26941746 THE BASICS: GRAPH PAPER
The heart then repolarises (or refills) in time for the next stimulus and contraction.
Atrial Fibrillation BRIEFLY, HOW DOES THE HEART PUMP? The heart has four chambers. The upper chambers are called atria. One chamber is called an atrium, and the lower chambers are called ventricles. In
QRS Complexes. Fast & Easy ECGs A Self-Paced Learning Program
6 QRS Complexes Fast & Easy ECGs A Self-Paced Learning Program Q I A ECG Waveforms Normally the heart beats in a regular, rhythmic fashion producing a P wave, QRS complex and T wave I Step 4 of ECG Analysis
Evaluation copy. Analyzing the Heart with EKG. Computer
Analyzing the Heart with EKG Computer An electrocardiogram (ECG or EKG) is a graphical recording of the electrical events occurring within the heart. In a healthy heart there is a natural pacemaker in
The P Wave: Indicator of Atrial Enlargement
Marquette University e-publications@marquette Physician Assistant Studies Faculty Research and Publications Health Sciences, College of 8-12-2010 The P Wave: Indicator of Atrial Enlargement Patrick Loftis
12-Lead EKG Interpretation. Judith M. Haluka BS, RCIS, EMT-P
12-Lead EKG Interpretation Judith M. Haluka BS, RCIS, EMT-P ECG Grid Left to Right = Time/duration Vertical measure of voltage (amplitude) Expressed in mm P-Wave Depolarization of atrial muscle Low voltage
Banner Staff Service ECG Study Guide
Banner Staff Service ECG Study Guide Edited by Larry H. Lybbert, MS, RN Table of Contents ECG STUDY GUIDE... 3 ECG INTERPRETATION BASICS... 4 EKG GRAPH PAPER...4 RATE MEASUREMENT...9 The Six Second Method...9
Electrodes placed on the body s surface can detect electrical activity, APPLIED ANATOMY AND PHYSIOLOGY. Circulatory system
4 READING AND INTERPRETING THE ELECTROCARDIOGRAM Electrodes placed on the body s surface can detect electrical activity, which occurs in the heart. The recording of these electrical events comprises an
Activity 4.2.3: EKG. Introduction. Equipment. Procedure
Activity 4.2.3: EKG The following is used with permission of Vernier Software and Technology. This activity is based on the experiment Analyzing the Heart with EKG from the book Human Physiology with Vernier,
Electrophysiology Introduction, Basics. The Myocardial Cell. Chapter 1- Thaler
Electrophysiology Introduction, Basics Chapter 1- Thaler The Myocardial Cell Syncytium Resting state Polarized negative Membrane pump Depolarization fundamental electrical event of the heart Repolarization
Basics of Pacing. Ruth Hickling, RN-BSN Tasha Conley, RN-BSN
Basics of Pacing Ruth Hickling, RN-BSN Tasha Conley, RN-BSN The Cardiac Conduction System Cardiac Conduction System Review Normal Conduction Conduction QRS QRS Complex Complex RR PP ST ST segment segment
The science of medicine. The compassion to heal.
A PATIENT S GUIDE TO ELECTROPHYSIOLOGY STUDIES OF THE HEART The science of medicine. The compassion to heal. This teaching booklet is designed to introduce you to electrophysiology studies of the heart.
the basics Perfect Heart Institue, Piyavate Hospital
ECG INTERPRETATION: the basics Damrong Sukitpunyaroj MD Damrong Sukitpunyaroj, MD Perfect Heart Institue, Piyavate Hospital Overview Conduction Pathways Systematic Interpretation Common abnormalities in
Novartis Gilenya FDO Program Clinical Protocol and Highlights from Prescribing Information (PI)
Novartis Gilenya FDO Program Clinical Protocol and Highlights from Prescribing Information (PI) Highlights from Prescribing Information - the link to the full text PI is as follows: http://www.pharma.us.novartis.com/product/pi/pdf/gilenya.pdf
Understanding the Electrocardiogram. David C. Kasarda M.D. FAAEM St. Luke s Hospital, Bethlehem
Understanding the Electrocardiogram David C. Kasarda M.D. FAAEM St. Luke s Hospital, Bethlehem Overview 1. History 2. Review of the conduction system 3. EKG: Electrodes and Leads 4. EKG: Waves and Intervals
Electrocardiography Review and the Normal EKG Response to Exercise
Electrocardiography Review and the Normal EKG Response to Exercise Cardiac Anatomy Electrical Pathways in the Heart Which valves are the a-v valves? Closure of the a-v valves is associated with which heart
Cardioversion for. Atrial Fibrillation. Your Heart s Electrical System Cardioversion Living with Atrial Fibrillation
Cardioversion for Atrial Fibrillation Your Heart s Electrical System Cardioversion Living with Atrial Fibrillation When You Have Atrial Fibrillation You ve been told you have a heart condition called atrial
Tachyarrhythmias (fast heart rhythms)
Patient information factsheet Tachyarrhythmias (fast heart rhythms) The normal electrical system of the heart The heart has its own electrical conduction system. The conduction system sends signals throughout
Systematic Approach to 12 Lead EKG Interpretation
Systematic Approach to 12 Lead EKG Interpretation Maureen Knechtel MPAS, PA-C Wellmont CVA Heart Institute Disclosure Statement of Financial Interest I, Maureen Knechtel, do not have a financial interest/arrangement
Exchange solutes and water with cells of the body
Chapter 8 Heart and Blood Vessels Three Types of Blood Vessels Transport Blood Arteries Carry blood away from the heart Transport blood under high pressure Capillaries Exchange solutes and water with cells
Lecture Outline. Cardiovascular Physiology. Cardiovascular System Function. Functional Anatomy of the Heart
Lecture Outline Cardiovascular Physiology Cardiac Output Controls & Blood Pressure Cardiovascular System Function Functional components of the cardiovascular system: Heart Blood Vessels Blood General functions
HTEC 91. Topic for Today: Atrial Rhythms. NSR with PAC. Nonconducted PAC. Nonconducted PAC. Premature Atrial Contractions (PACs)
HTEC 91 Medical Office Diagnostic Tests Week 4 Topic for Today: Atrial Rhythms PACs: Premature Atrial Contractions PAT: Paroxysmal Atrial Tachycardia AF: Atrial Fibrillation Atrial Flutter Premature Atrial
Normal Sinus Rhythm. Sinus Bradycardia. Sinus Tachycardia. Rhythm ECG Characteristics Example (NSR) & consistent. & consistent.
Normal Sinus Rhythm (NSR) Rate: 60-100 per minute Rhythm: R- R = P waves: Upright, similar P-R: 0.12-0.20 second & consistent P:qRs: 1P:1qRs Sinus Tachycardia Exercise Hypovolemia Medications Fever Hypoxia
How to read the ECG in athletes: distinguishing normal form abnormal
How to read the ECG in athletes: distinguishing normal form abnormal Antonio Pelliccia, MD Institute of Sport Medicine and Science www.antoniopelliccia.it Cardiac adaptations to Rowing Vagotonia Sinus
Bradycardia CHAPTER 12 CODE SCENARIO
Senecal-12.qxd 14/04/2005 09:44 AM Page 69 CHAPTER 12 Bradycardia CODE SCENARIO A code is called for a 78-year-old man who was admitted to the hospital for syncope of unknown etiology. He was resting comfortably
BIPOLAR LIMB LEADS UNIPOLAR LIMB LEADS PRECORDIAL (UNIPOLAR) LEADS VIEW OF EACH LEAD INDICATIVE ECG CHANGES
BIPOLAR LIMB LEADS Have both a distinctive positive and negative pole. Lead I LA (positive) RA (negative) Lead II LL (positive) RA (negative) Lead III LL (positive) LA (negative) UNIPOLAR LIMB LEADS Have
Acquired, Drug-Induced Long QT Syndrome
Acquired, Drug-Induced Long QT Syndrome A Guide for Patients and Health Care Providers Sudden Arrhythmia Death Syndromes (SADS) Foundation 508 E. South Temple, Suite 202 Salt Lake City, Utah 84102 800-STOP
Atrial & Junctional Dysrhythmias
Atrial & Junctional Dysrhythmias Atrial & Junctional Dysrhythmias Atrial Premature Atrial Complex Wandering Atrial Pacemaker Atrial Tachycardia (ectopic) Multifocal Atrial Tachycardia Atrial Flutter Atrial
ACLS PHARMACOLOGY 2011 Guidelines
ACLS PHARMACOLOGY 2011 Guidelines ADENOSINE Narrow complex tachycardias or wide complex tachycardias that may be supraventricular in nature. It is effective in treating 90% of the reentry arrhythmias.
An ECG Primer. Quick Look. I saw it, but I did not realize it. Elizabeth Peabody
4 An ECG Primer Quick Look Cardiac Monitoring System - p. 64 ECG Paper - p. 73 Lead Polarity and Vectors - p. 77 Basic ECG Components - p. 79 Heart Rate and Pulse Rate - p. 91 Summary - p. 94 Chapter Quiz
Introduction to Electrophysiology. Wm. W. Barrington, MD, FACC University of Pittsburgh Medical Center
Introduction to Electrophysiology Wm. W. Barrington, MD, FACC University of Pittsburgh Medical Center Objectives Indications for EP Study How do we do the study Normal recordings Abnormal Recordings Limitations
Catheter Ablation. A Guided Approach for Treating Atrial Arrhythmias
Catheter Ablation A Guided Approach for Treating Atrial Arrhythmias A P A T I E N T H A N D B O O K This brochure will provide an overview of atrial arrhythmias (heart rhythm problems affecting the upper
HEART HEALTH WEEK 3 SUPPLEMENT. A Beginner s Guide to Cardiovascular Disease HEART FAILURE. Relatively mild, symptoms with intense exercise
WEEK 3 SUPPLEMENT HEART HEALTH A Beginner s Guide to Cardiovascular Disease HEART FAILURE Heart failure can be defined as the failing (insufficiency) of the heart as a mechanical pump due to either acute
Current Management of Atrial Fibrillation DISCLOSURES. Heart Beat Anatomy. I have no financial conflicts to disclose
Current Management of Atrial Fibrillation Mary Macklin, MSN, APRN Concord Hospital Cardiac Associates DISCLOSURES I have no financial conflicts to disclose Book Women: Fit at Fifty. A Guide to Living Long.
Distance Learning Program Anatomy of the Human Heart/Pig Heart Dissection Middle School/ High School
Distance Learning Program Anatomy of the Human Heart/Pig Heart Dissection Middle School/ High School This guide is for middle and high school students participating in AIMS Anatomy of the Human Heart and
Monitoring EKG. Evaluation copy
Monitoring EKG Computer 28 An electrocardiogram, or EKG, is a graphical recording of the electrical events occurring within the heart. A typical EKG tracing consists of five identifiable deflections. Each
Atrial Fibrillation (AF) March, 2013
Atrial Fibrillation (AF) March, 2013 This handout is meant to help with discussions about the condition, and it is not a complete discussion of AF. We hope it will complement your appointment with one
Chapter 20: The Cardiovascular System: The Heart
Chapter 20: The Cardiovascular System: The Heart Chapter Objectives ANATOMY OF THE HEART 1. Describe the location and orientation of the heart within the thorax and mediastinal cavity. 2. Describe the
Diagnosis Code Crosswalk : ICD-9-CM to ICD-10-CM Cardiac Rhythm and Heart Failure Diagnoses
Diagnosis Code Crosswalk : to 402.01 Hypertensive heart disease, malignant, with heart failure 402.11 Hypertensive heart disease, benign, with heart failure 402.91 Hypertensive heart disease, unspecified,
Atrioventricular (AV) node ablation
Patient information factsheet Atrioventricular (AV) node ablation The normal electrical system of the heart The heart has its own electrical conduction system. The conduction system sends signals throughout
Interpreting a rhythm strip
3 Interpreting a rhythm strip Just the facts In this chapter, you ll learn: the components of an ECG complex and their significance and variations techniques for calculating the rate and rhythm of an ECG
RAPID INTERPRETATION OF. EKG s
Personal Quick Reference Sheets 333 (pages 333 to 346) There is no need to remove these reference pages from your book. To download and print them in full color, go to: www.themdsite.com Reference Sheets
Atrial Fibrillation Management Across the Spectrum of Illness
Disclosures Atrial Fibrillation Management Across the Spectrum of Illness NONE Barbara Birriel, MSN, ACNP-BC, FCCM The Pennsylvania State University Objectives AF Discuss the pathophysiology, diagnosis,
Biology 347 General Physiology Lab Advanced Cardiac Functions ECG Leads and Einthoven s Triangle
Biology 347 General Physiology Lab Advanced Cardiac Functions ECG Leads and Einthoven s Triangle Objectives Students will record a six-lead ECG from a resting subject and determine the QRS axis of the
Electrocardiogram and Heart Sounds
Electrocardiogram and Heart Sounds An introduction to the recording and analysis of electrocardiograms, and the sounds of the heart. Written by Staff of ADInstruments Introduction The beating of the heart
ANNE ARUNDEL MEDICAL CENTER CRITICAL CARE MEDICATION MANUAL DEPARTMENT OF NURSING AND PHARMACY. Guidelines for Use of Intravenous Isoproterenol
ANNE ARUNDEL MEDICAL CENTER CRITICAL CARE MEDICATION MANUAL DEPARTMENT OF NURSING AND PHARMACY Guidelines for Use of Intravenous Isoproterenol Major Indications Status Asthmaticus As a last resort for
Signal-averaged electrocardiography late potentials
SIGNAL AVERAGED ECG INTRODUCTION Signal-averaged electrocardiography (SAECG) is a special electrocardiographic technique, in which multiple electric signals from the heart are averaged to remove interference
Welcome to Vibrationdata
Welcome to Vibrationdata Acoustics Shock Vibration Signal Processing December 2004 Newsletter Ni hao Feature Articles One of my goals is to measure a wide variety of oscillating signals. In some sense,
COVERAGE GUIDANCE: ABLATION FOR ATRIAL FIBRILLATION
COVERAGE GUIDANCE: ABLATION FOR ATRIAL FIBRILLATION Question: How should the EGBS Coverage Guidance regarding ablation for atrial fibrillation be applied to the Prioritized List? Question source: Evidence
Anatomi & Fysiologi 060301. The cardiovascular system (chapter 20) The circulation system transports; What the heart can do;
The cardiovascular system consists of; The cardiovascular system (chapter 20) Principles of Anatomy & Physiology 2009 Blood 2 separate pumps (heart) Many blood vessels with varying diameter and elasticity
Basic Cardiac Rhythms Identification and Response
Basic Cardiac Rhythms Identification and Response Module 1 ANATOMY, PHYSIOLOGY, & ELECTRICAL CONDUCTION Objectives Describe the normal cardiac anatomy and physiology and normal electrical conduction through
Electrophysiology study (EPS)
Patient information factsheet Electrophysiology study (EPS) The normal electrical system of the heart The heart has its own electrical conduction system. The conduction system sends signals throughout
Treating AF: The Newest Recommendations. CardioCase presentation. Ethel s Case. Wayne Warnica, MD, FACC, FACP, FRCPC
Treating AF: The Newest Recommendations Wayne Warnica, MD, FACC, FACP, FRCPC CardioCase presentation Ethel s Case Ethel, 73, presents with rapid heart beating and mild chest discomfort. In the ED, ECG
ECG made extra easy. medics.cc
ElectroCardioGraphyraphy ECG made extra easy Overview Objectives for this tutorial What is an ECG? Overview of performing electrocardiography on a patient Simple physiology Interpreting the ECG Objectives
Section Four: Pulmonary Artery Waveform Interpretation
Section Four: Pulmonary Artery Waveform Interpretation All hemodynamic pressures and waveforms are generated by pressure changes in the heart caused by myocardial contraction (systole) and relaxation/filling
Heart and Vascular System Practice Questions
Heart and Vascular System Practice Questions Student: 1. The pulmonary veins are unusual as veins because they are transporting. A. oxygenated blood B. de-oxygenated blood C. high fat blood D. nutrient-rich
The Heart Rhythm Charity
The Heart Rhythm Charity Promoting better understanding, diagnosis, treatment and quality of life for individuals with cardiac arrhythmias Registered Charity No. 1107496 2006 Bradycardia (Slow Heart Rhythm)
Potential Causes of Sudden Cardiac Arrest in Children
Potential Causes of Sudden Cardiac Arrest in Children Project S.A.V.E. When sudden death occurs in children, adolescents and younger adults, heart abnormalities are likely causes. These conditions are
INFORMATION FOR PATIENTS AND FAMILIES A Patient s Guide to Living with Atrial Fibrillation
INFORMATION FOR PATIENTS AND FAMILIES A Patient s Guide to Living with Atrial Fibrillation 30 Bond Street, Toronto, ON M5B 1W8 Canada 416.864.6060 stmichaelshospital.com Form No. XXXXX Dev. XX/XXXX GOALS
22 Arrhythmias. C. Scharf and F. Duru. Siegenthaler, Differential Diagnosis in Internal Medicine (ISBN9783131421418), 2007 Georg Thieme Verlag
22 22 Arrhythmias C. Scharf and F. Duru 22 712 Arrhythmias 22.1 Differential Diagnosis of Arrhythmias 714 Medical History 714 Clinical Examination 714 Electrocardiogram (ECG) 715 Additional Tools for the
PATIENT INFORMATION GUIDE TO ATRIAL FIBRILLATION
PATIENT INFORMATION GUIDE TO ATRIAL FIBRILLATION A Comprehensive Resource from the Heart Rhythm Society AF 360 provides a single, trusted resource for the most comprehensive and relevant information and
GUIDE TO ATRIAL FIBRILLATION
PATIENT INFORMATION GUIDE TO ATRIAL FIBRILLATION Atrial Fibrillation (AF) Atrial Flutter (AFL) Rate and Rhythm Control Stroke Prevention This document is endorsed by: A Comprehensive Resource from the
DEPARTMENT OF HEALTH AND HUMAN SERVICES Centers for Medicare & Medicaid Services
DEPARTMENT OF HEALTH AND HUMAN SERVICES Centers for Medicare & Medicaid Services NEW product from the Medicare Learning Network (MLN) Provider Compliance Tips for Computed Tomography (CT) Scans Podcast,
TOP 5. The term cardiac arrhythmia encompasses all cardiac. Arrhythmias in Dogs & Cats. Sinus Arrhythmia. TOP 5 Arrhythmias Seen in Dogs & Cats
Top 5 ardiology Peer reviewed TOP 5 rrhythmias in Dogs & ats shley Jones, DVM mara Estrada, DVM, DVIM (ardiology) University of Florida The term cardiac arrhythmia encompasses all cardiac rhythms other
ACLS RHYTHM TEST. 2. A 74-year-old woman with chest pain. Blood pressure 192/90 and rates her pain 9/10.
ACLS RHYTHM TEST Name Date Choose the best answer for each of the following questions. Each of the following strips is 6 seconds in length. 1. Identify the following rhythm a. Sinus bradycardia with 2
Medical management of CHF: A New Class of Medication. Al Timothy, M.D. Cardiovascular Institute of the South
Medical management of CHF: A New Class of Medication Al Timothy, M.D. Cardiovascular Institute of the South Disclosures Speakers Bureau for Amgen Background Chronic systolic congestive heart failure remains
Advanced Cardiovascular Life Support Case Scenarios
Advanced Cardiovascular Life Support Case Scenarios ACLS Respiratory Arrest Case Out-of-Hospital Scenario You are a paramedic and respond to the scene of a possible cardiac arrest. A young man lies motionless
Table of Contents Error! Bookmark not defined.
Table of Contents EKG TRACING...1 Figure 1 - EKG Tracing... Error! Bookmark not defined. STEP 1...1 Rate... 1 Figure 2 - Determining the Rate... 1 Step 2...2 Rhythm... 2 Figure 3 - Determining the Rhythm
GUIDELINE 11.9 MANAGING ACUTE DYSRHYTHMIAS. (To be read in conjunction with Guideline 11.7 Post-Resuscitation Therapy in Adult Advanced Life Support)
AUSTRALIAN RESUSCITATION COUNCIL GUIDELINE 11.9 MANAGING ACUTE DYSRHYTHMIAS (To be read in conjunction with Guideline 11.7 Post-Resuscitation Therapy in Adult Advanced Life Support) The term cardiac arrhythmia
Atrial Fibrillation An update on diagnosis and management
Dr Arvind Vasudeva Consultant Cardiologist Atrial Fibrillation An update on diagnosis and management Atrial fibrillation (AF) remains the commonest disturbance of cardiac rhythm seen in clinical practice.
Atrial Fibrillation: The heart of the matter
Atrial Fibrillation: The heart of the matter This booklet has been written especially for people with atrial fibrillation (AF), a heart condition often described as an irregular heartbeat (also known
Quiz 4 Arrhythmias summary statistics and question answers
1 Quiz 4 Arrhythmias summary statistics and question answers The correct answers to questions are indicated by *. All students were awarded 2 points for question #2 due to no appropriate responses for
Advanced EKG Interpretation
Advanced EKG Interpretation JUNCTIONAL RHYTHMS AND NURSING INTERVENTIONS Objectives Identify specific cardiac dysrhythmias Describe appropriate nursing interventions for specific dysrhythmias Junctional
ST Segment Elevation Nothing is ever as hard (or easy) as it looks
ST Segment Elevation Nothing is ever as hard (or easy) as it looks Cameron Guild, MD Division of Cardiology University of Mississippi Medical Center February 17, 2012 Objectives 1. Describe the electrical
Visited 9/14/2011. What is Atrial Fibrillation? What you need to know about Atrial Fibrillation. The Normal Heart Rhythm. 1 of 7 9/14/2011 10:50 AM
1 of 7 9/14/2011 10:50 AM Current URL: What you need to know about Atrial Fibrillation What is atrial fibrillation? What causes atrial fibrillation? How is atrial fibrillation diagnosed? What are the dangers
Recurrent AF: Choosing the Right Medication.
In the name of God Shiraz E-Medical Journal Vol. 11, No. 3, July 2010 http://semj.sums.ac.ir/vol11/jul2010/89015.htm Recurrent AF: Choosing the Right Medication. Basamad Z. * Assistant Professor, Department
Introduction. What is syncope?
Syncope Introduction What is syncope? Syncope (SING-kuh-pee) is a medical term for fainting. When you faint, your brain is not receiving enough blood and oxygen, so you lose consciousness temporarily.
Atrial Fibrillation. Information for you, and your family, whänau and friends. Published by the New Zealand Guidelines Group
Atrial Fibrillation Information for you, and your family, whänau and friends Published by the New Zealand Guidelines Group CONTENTS Introduction 1 The heart 2 What is atrial fibrillation? 3 How common
Atrial Fibrillation Centre
About this guide We have prepared this guide to help you to: learn about atrial fibrillation manage atrial fibrillation and reduce the risk of stroke find out about medicines and other treatment options
Radiofrequency Ablation for Atrial Fibrillation. A Guide for Adults
Radiofrequency Ablation for Atrial Fibrillation A Guide for Adults Fast Facts n There are different ways to treat atrial fibrillation (A-fib). One kind of treatment involves putting the heart back into
NAME OF THE HOSPITAL: 1. Coronary Balloon Angioplasty: M7F1.1/ Angioplasty with Stent(PTCA with Stent): M7F1.3
1. Coronary Balloon Angioplasty: M7F1.1/ Angioplasty with Stent(PTCA with Stent): M7F1.3 1. Name of the Procedure: Coronary Balloon Angioplasty 2. Select the Indication from the drop down of various indications
The Basics of 12 Lead EKG s
EMS Solutions Presents The Basics of 12 Lead EKG s NOTICE: You DO NOT Have the Right to Reprint or Resell this Publication. However, you MAY give this report away, provided you do not change or alter the
PRO-CPR. 2015 Guidelines: PALS Algorithm Overview. (Non-AHA supplementary precourse material)
PRO-CPR 2015 Guidelines: PALS Algorithm Overview (Non-AHA supplementary precourse material) Please reference Circulation (from our website), the ECC Handbook, or the 2015 ACLS Course Manual for correct
How To Understand What You Know
Heart Disorders Glossary ABG (Arterial Blood Gas) Test: A test that measures how much oxygen and carbon dioxide are in the blood. Anemia: A condition in which there are low levels of red blood cells in
Atrial Fibrillation (AF) Explained
James Paget University Hospitals NHS Foundation Trust Atrial Fibrillation (AF) Explained Patient Information Contents What are the symptoms of atrial fibrillation (AF)? 3 Normal heartbeat 4 How common
PSIO 603/BME 511 1 Dr. Janis Burt February 19, 2007 MRB 422; 626-6833 [email protected]. MUSCLE EXCITABILITY - Ventricle
SIO 63/BME 511 1 Dr. Janis Burt February 19, 27 MRB 422; 626-6833 MUSCLE EXCITABILITY - Ventricle READING: Boron & Boulpaep pages: 483-57 OBJECTIVES: 1. Draw a picture of the heart in vertical (frontal
What to Know About. Atrial Fibrillation
Atrial Fibrillation What to Know About Atrial Fibrillation Understanding Afib Atrial fibrillation, or Afib, is a condition in which the heart beats irregularly speeding up or slowing down, or beating too
