Acute Pancreatitis, Diagnosis and Management



Similar documents
Acute Pancreatitis. Questionnaire. if yes: amount (cigarettes/day): since when (year): Drug consumption: yes / no if yes: type of drug:. amount:.

Practice Guidelines in Acute Pancreatitis

Pancréatite Aiguë Prise en charge en L Bühler. Service de Chirurgie Viscérale et Transplantation Département de Chirurgie HUG

American College of Gastroenterology Guideline: Management of Acute Pancreatitis

The Sepsis Puzzle: Identification, Monitoring and Early Goal Directed Therapy

Suffolk County Community College School of Nursing NUR 133 ADULT NURSING I

Decreasing Sepsis Mortality at the University of Colorado Hospital

The Initial and 24 h (After the Patient Rehabilitation) Deficit of Arterial Blood Gases as Predictors of Patients Outcome

Lynda Richardson, RN, BSN Sepsis/Septic Shock Abstractor. No disclosures

Core Measures SEPSIS UPDATES

Diabetic Ketoacidosis: When Sugar Isn t Sweet!!!

NUTRITION IN LIVER DISEASES

SE5h, Sepsis Education.pdf. Surviving Sepsis

Amylase and Lipase Tests

Colocutaneous Fistula. Disclosures

28 Year-old Male w/uncontrolled Type 2 Diabetes, Bipolar Disorder Presents with Epigastric Pain. Jess Hwang 9/27/12

Open the Flood Gates Urinary Obstruction and Kidney Stones. Dr. Jeffrey Rosenberg Dr. Emilio Lastarria Dr. Richard Kasulke

Georgia Northwestern Technical College Practical Nursing Program CLINICAL DAILY ASSESSMENT WORKSHEET FOR MODULES I-IV STUDENT: CLINICAL INSTRUCTOR:

Cardiovascular diseases. pathology

Diseases of peritoneum Lect. Al Qassim University, Faculty of Medicine Phase II Year III, CMD 332 Pathology Department 31-32

Acute Abdominal Pain following Bariatric Surgery. Disclosure. Objectives 8/17/2015. I have nothing to disclose

Steven B. Goldin, MD, PhD University of South Florida Dimitrios Stefanidis, MD, PhD

Malpractice and the Infectious Disease Any Physician WHAT YOU SHOULD KNOW! Why this talk? Why me?

Early Colonoscopy in Patients with Acute Diverticulitis Simon Bar-Meir, M.D.

John Gasman, MD Alec Jamieson, RN, MSN Kim Clifforth, RN, BSN, MSN, CNS Thomas T. Lam, MD. June 18, 2013

Endoscopy and infection: Prevention of infection during endoscopy Treatment of infection by endoscopy. M. Arvanitakis SRBG June 2009

Emergencies in Post- Bariatric Surgery Patients

Pathway for the Management of Acute Gallstone Diseases

Approach to Abnormal Liver Tests

TOTAL PARENTERAL NUTRITION (TPN) Revised January 2013

To Whipple or Not to Whipple, that is the Question: Evaluating the Resectability of Pancreatic Adenocarcinoma

Liver, Gallbladder and Pancreas diseases. Premed 2 Pathophysiology

DAYTON CHILDREN S HOSPITAL CLINICAL PRACTICE GUIDELINES

What to do with abnormal LFTs? Andrew M Smith Hepatobiliary Surgeon

Bile Duct Diseases and Problems

Subject: Severe Sepsis/Septic Shock Published Date: August 9, 2013 Scope: Hospital Wide Original Creation Date: August 9, 2013

Omega-3 fatty acids improve the diagnosis-related clinical outcome. Critical Care Medicine April 2006;34(4):972-9

Patterns of abnormal LFTs and their differential diagnosis

Quiz 5 Heart Failure scores (n=163)

Dehydration in Pediatrics. Dr. Erin Killorn Pediatric Emergency physician February 19 th, 2015

Evaluation of Liver Function tests in Primary Care. Abid Suddle Institute of Liver Studies, KCH

Head Injury. Dr Sally McCarthy Medical Director ECI

ABThera Open Abdomen Negative Pressure Therapy for Active Abdominal Therapy. Case Series

Interpretation of Laboratory Values

Sepsis Reassess patient Monitor and maintain respiratory/ hemodynamic status

ACID-BASE DISORDER. Presenter: NURUL ATIQAH AWANG LAH Preceptor: PN. KHAIRUL BARIAH JOHAN

Renal Cysts What should I do now?

EVIDENCE BASED TREATMENT OF CROHN S DISEASE. Dr E Ndabaneze

Hepatocellular Carcinoma (HCC)

6/3/2011. High Prevalence and Incidence. Low back pain is 5 th most common reason for all physician office visits in the U.S.

Obesity Affects Quality of Life

Module 9: Diseases of the Endocrine System and Nutritional Disorders Exercises

2002 burns responsible for 322,000 deaths world wide. aboriginal community in NA Most burns occur in the urban environment

Adult CCRN/CCRN E/CCRN K Certification Review Course: Integumentary and Musculoskeletal

OMG my LFT s! How to Interpret and Use Them. OMG my LFT s! OMG my LFT s!

La pancréatite aiguë aux urgences

INR: RUPTURED ANEURYSM: POST EMBOLIZATION Patient Identification Page 1 of 5. Allergies: Weight: kg Diagnosis:

Early Warning Scores (EWS) Clinical Sessions 2011 By Bhavin Doshi

Oxygenation. Chapter 21. Anatomy and Physiology of Breathing. Anatomy and Physiology of Breathing*

Metabolic Syndrome Overview: Easy Living, Bitter Harvest. Sabrina Gill MD MPH FRCPC Caroline Stigant MD FRCPC BC Nephrology Days, October 2007

Sepsis: Identification and Treatment

Recommendations: Other Supportive Therapy of Severe Sepsis*

RGN JOY LAUDE WATFORD GENERAL HOSPITAL, ENGLAND

EXECUTIVE BLOOD WORK PANEL

Diabetes Expert Witness on: Diabetic Hypoglycemia in Nursing Homes

Marilyn Borkgren-Okonek, APN, CCNS, RN, MS Suburban Lung Associates, S.C. Elk Grove Village, IL

Understanding Mortality Statistics: The Importance of Cause-of-Death Certification and Coding

Management of Adverse Drug Reactions in Tuberculosis. Anju Budhwani, MD

POAC CLINICAL GUIDELINE

V: Infusion Therapy. Alberta Licensed Practical Nurses Competency Profile 217

Nutr 341: Medical Nutrition Therapy: A Case Study Approach 3 rd ed. Case 32 Esophageal Cancer Treated with Surgery and Radiation

The child with abnormal liver function tests

APPENDIX B SAMPLE PEDIATRIC CRITICAL CARE NURSE PRACTITIONER GOALS AND OBJECTIVES

Inflammation and Healing. Review of Normal Defenses. Review of Normal Capillary Exchange. BIO 375 Pathophysiology

ETIOLOGIC CLASSIFICATION. Type I diabetes Type II diabetes

Acute abdominal conditions Key Points

A: Nursing Knowledge. Alberta Licensed Practical Nurses Competency Profile 1

Medical Direction and Practices Board WHITE PAPER

Advanced Practice Provider Academy

Diabetic Emergencies. David Hill, D.O.

Nursing Care and Considerations for Patients with Atrial Fibrillation. Kris Kinghorn RN, MSN, ANP-BC

Acid-Base Balance and the Anion Gap

Case Number: RT (M) Potential Audiences: Intent Doctor, Oncology Special Nurse, Resident Doctor

Arterial Blood Gas Case Questions and Answers

High Blood Cholesterol

KIDNEY FUNCTION RELATION TO SIZE OF THE TUMOR IN RENAL CELL CANCINOMA

Disability Evaluation Under Social Security

Open Ventral Hernia Repair

Definition, Prevalence, Pathophysiology and Complications of CKD. JM Krzesinski CHU Liège-ULg Core curriculum Nephrology September 28 th 2013

V: Infusion Therapy. College of Licensed Practical Nurses of Alberta, Competency Profile for LPNs, 3rd Ed. 181

Transcription:

Acute Pancreatitis, Diagnosis and Management Tedra D. Gray, BSN, MS, ACNP-BC Sinai Health Systems Internal Medicine Department Division of Gastroenterology

Acute Pancreatitis Annual incidence of pancreatitis 280,000 hospitalizations per year Overall mortality of hospitalized patients is approximately 10% whereas the subset with severe acute pancreatitis is as high as 30% www.uptodate.com 2011

Acute Pancreatitis - Pathophysiology Induction of acute pancreatitis due to intraacinar activation proteolytic enzymes, which ultimately leads to an autodigestive injury to the gland Activated pancreatic enzymes, microcirculatory impairment, and release of inflammatory mediators lead to rapid worsening of pancreatic damage and necrosis Revision of Atlanta Classification 2009

Acute Pancreatitis - International Atlanta Symposium Definition Acute inflammatory process of the pancreas that may also involve peripancreatic tissue and / or remote organ systems Includes organ failure- shock, pulmonary insufficiency, renal failure, gastrointestinal bleeding Local complications pancreatic necrosis, abscess, pseudocyst

Acute Pancreatitis - Definition Inflammatory condition of the pancreas requires two of the following three features: 1) abdominal pain suggestive strongly of acute pancreatitis 2) serum amylase and/or lipase 3 X ULN 3) characteristic findings of acute pancreatitis on transabdominal US or CECT Revision of Atlanta Symposium 2009

Acute Pancreatitis - Overview Diagnosis Etiology H&P, Labs, Imaging Cholelitiasis, Alcohol, Anatomical Varients, etc. Manage Complications Pancreatic Necrosis, Infections, Pseudocysts General Supportive Care Clinical Approach to Treatment

Acute Pancreatitis Clinical Features N/V & ABD pain radiating to the scapula area /back relieved by leaning forward occurs in 40 70 % of pt. Grey Turner flank discoloration Cullen sign - peri umbilical discoloration Amylase & Lipase Gastro 2007;132:2022-2044

Acute Pancreatitis - Labs Amylase 40% of serum from pancreas thus note other causes Sen. 72%, Spec 99% Levels for Dx 3X ULN Lipase level 3X ULN Sen. 100%, Spec 96% Elevated longer Varied Causes Frequency Dx??? Gastro 2007;132:2022-2044

Acute Pancreatitis - Phases Interstitial (1 st phase): Onset within 1 st week Severity R/T organ failure and response to SIRS Mild Mod-Severe Organ Failure<48hrs Necrotizing Severe (2 nd Phase): Organ Failure >48 hrs Protracted course R/T necrotizing process

Acute Pancreatitis - Imaging Ultrasound Identifies cholelithiasis and the presence of choledocholithiasis Sensitivity of ~ 70% Contrast Enhanced Comp Tomography Timing Used to confirm diagnosis but NOT needed Gastro 2007; 132: 2022-2044

Acute Pancreatitis - Imaging What does a CT Scan Offer? Excludes alternative diagnosis, determine severity, identify complications What does it show? Isolated, or diffuse enlargement or peripancreatic stranding and fluid collections Necrosis apparent 3 days after ONSET of disease and seen as absence of enhancing of parenchyma after IV contrast

Acute Pancreatitis - Imaging CECT showing peripancreatic and retroperitoneal edema (large arrows) and stranding. The pancreas itself (small arrow) appears relatively normal.

Acute Pancreatitis - MRI Lack nephrotoxicity Sensitivity Visualization of biliary and pancreatic ductal system Limitiations

Acute Pancreatitis - Etiology Cholelithiasis Alcohol Anatomic Variants Metabolic Disorders Drugs Trauma (Blunt vs. Post ERCP) Autoimmune Infections Genetic

Severity How bad is it? Demographics & Lab data Organ Failure Imaging

Acute Pancreatitis - Severity Ranson Criteria ADMISSION Age >55 (70) WBC >16 K(18K) Glucose >200 (220) LDH >350 (400) AST >250 48 HOURS: Drop in HCT >10%, > 6L (4) Fluid needs Ca <8 mg/dl PaO2 <60 mm Hg BUN increase > 5 mg/dl (2), BD > 4mmol/L (6) Gastro 2007; 132: 2022-2044

Acute Pancreatitis -Severity Calculate on admission & at 24 and 48 hours; Admit score > 7 predict severe pancreatitis sensitivity 65% spec 76%, PPV 43% and NPV 93% Score at 48 hours predicts severe pancreatitis 76% and spec 84% with PPV 54% and NPV 93% APACHE II Hx of severe organ insuff. of immunoco mpromise d or post op state Age, GCS, Temp, MAP, HR, A-a & FiO2, Arterial ph, Serum Co2, Na, K, Creat ARF, WBC Gastro 2007; 132: 2022-2044

Marshall Scoring System ORGAN SYSTEM Resp (PO2/FIO2) Renal (Creat) CV: (SBP) 0 1 2 3 4 >400 301-400 201-300 101-200 <101 <1.4 1.4-1.8 1.9-3.6 3.6-4.9 >4.9 >90 <90 FR <90 NFR < 90 ph<7.3 <90 ph<7.2

Acute Pancreatitis CT Severity Index Pancreatic necrosis is a major negative prognostic factor Large retrospective analysis of 268 patients found that a CT severity index >5 significantly correlated with death, prolonged hospital stay and need for necrosectomy Gastro 2007; 132: 2022-2044

Acute Pancreatitis - CT Severity Index Components 0 - Normal 1 - Focal/diffuse enlargement 2 - Instrinsic abnormalities and inflammation in peripancreatic fat 3 - Single, ill-defined fluid collection or phlegmon 4 - Two or more poorly defined collections, presence of gas in or adjacent to pancreas

Acute Pancreatitis - CT Severity Index Components 0 - No necrosis 2 - Less than or equal to 30% 4 - Greater than 30 and less than or equal to 50% 6 - Greater than 50%

Acute Pancreatitis - Severity Balthazar CT Score A Normal B Focal or diffuse enlargement of the pancreas C finding of B with peripancreatic inflammation D findings of C + a single fluid collection E Findings of C + 2 or more fluid collections and/or presence of gas in/or adjacent to pancreas

63 Acute Pancreatitis CT Severity Index Multiple studies with head to head comparison APACHE II outperformed Ranson s Criteria Balthazar CT Score suggested superior in predicting necrosis but less accurate in predicting organ failure Combining CT findings and APACHE II improved predictive outcome over APACHE II alone

Case Study # 1 52 yr old male PMHx: HTN, DM, Etoh Abuse, GIB 3/2013 and pancreatitis 4/2013 cc: 1 wk of sharp epigastric ABD pain with radiation to back, n/v VS: T 98.9, HR 142, B/P 150/97, RR 26 PE: Awake /Restless Lungs clear Distended ABD, mod tenderness, hypoactive BS. No ascities

Case Study # 1 Glucose 311 HCO3 14, K 3.5 Ca 7.7, lactate 9.1 BUN 17, Creat 0.77 LDH 754, Hct 41% Lipase 1,210 T. B. 2.2, AST 888, ALT 557, Alk phos 160 CT of ABD done without IV contrast due to IVP allergy; Ext peripancreatic inflammatroy changes with some free fluid, no gallstones

Acute Pancreatitis - Overview Diagnosis Etiology H&P, Labs, Imaging Alcohol Manage Complications Clinical Approach to Treatment

Acute Pancreatitis Etiology Alcohol Alcohol activates the pancreatic enzymes in the acniar cells which causes digestion of lysosomal enzymes that are thought to cause pancreatitis Accounts for ~30% of acute on chronic pancreatitis episodes

Acute Pancreatitis - Management Fluid Resuscitation Crystalloids/ LR Bolus 20ml/kg Goal of 0.5ml/kg of body weight urine output O2 requirements, acidosis, monitor ionized Ca, control glucose, Metabolic Imbalance Nutrition > 72 hours of NPO NPO vs PO Intake NJ vs. TPN Hydromorphone commonly used Pain Control

Case Study # 1 - Recs Hydration: cont @ 3ml/kg; Reassess Q 6-8 hrs Watch electrolytes & BUN i.e Ca, Mg, acid base balance Oxygen support PPI (Hx of GIB due to Mallory Weiss Tear) Antibiotics???

Case Study # 1 140 114 31 14.9 230 17.0 71 4.5 15 2.94 44.8 Ca 5.8, Lipase 744 MCV 87.6 Lactate 9.1 Bands 32 (Temp 103.9) ABG:pH 7.07, PcO2 53, PO2 108,Hco3 15.4,BE -15 Input: 5,856cc Output:150cc

Acute Pancreatitis - Management Adequate fluid resuscitation Correct electrolyte & metabolic imbalance Pain control Nutritional support

Case Study # 1 Pt intubated developed infiltrates on CXR 4 pressors started ; spiking fevers ABD distention worsened

Case Study # 1 What do you look at today? What would you do differently in your management and recommendation?

Acute Pancreatitis Organ Failure SHOCK (SBP< 90 mm Hg) Pulmonary Insufficiency (PaO2 <60 mm Hg) Renal Failure (creatinine >2 mg/dl) GIB > 500 ml of blood loss within 24 hours Pancreatic specific Issues; pseudosyst, abscess, parenchymal necrosis Gastro 2007; 132: 2022-2044

Case Study # 1 : Outcome LOS: 42 days ARDS/ Trach & PEG Repeat CT: Hypodense fluid collection 8.2 X 4.8 cm ant/inferior to pancreas.??? Phlegmon or early abscess IR: drain placed AKI : HD for 1mth Sent to rehab due to reconditioning

Acute Pancreatitis - Management Prophylactic antibiotic Pancreatic specific antibiotics introduced when at least 30% necrosis seen on CT Drug of choice

Acute Pancreatitis - Imaging Peripancreatic and retroperitoneal edema. Large non-enhancing areas of necrosis are visible in the body and neck of the pancreas (arrows).

Acute Pancreatitis - Complications Pancreatic Necrosis Best identified on CECT MRI/EUS to characterize content

Acute Pancreatitis - Complications Pancreatic Infections Worsen S/S 1-2 weeks after onset CECT presence of gas highly suggestive of diagnosis FNA; gram stain, Cx Rx; Antibiotics, CT guided percutaneous drainage, open surgical debridement with necrosectomy

Acute Pancreatitis Complications Pseudocyst Common in moderate & severe case Usually resolve in 6 weeks; reassess Size matters; <6cm monitor >6 cm drainage needed Monitor for s/s of obstruction, infection, rupture or bleeding Prior to treatment; confirm contents with EUS

Acute Pancreatitis - Well-defined fluid collection in the retroperitoneum (arrow) just below the level of the pancreas. Complications

Acute Pancreatitis Nutritional Support Clinical judgement for resumption of PO intake; key elements absent ileus and pain control Meta analysis of 6 randomized trials of TPN vs NJ feeds NJ feeds have reduced infection & need for surgery but no decrease in organ failure or mortality Gastro 2007; 132: 2022-2044

Acute Pancreatitis Conclusion 85 % of patients develop interstitial edematous pancreatitis 15% of patients develop necrotizing pancreatitis 1 in 5 will develop severe pancreatitis of which 20 30 % will die Gastro 2007; 132: 2022-2044

Acute Pancreatitis MILD Resuscitation & SEVERE APACHE >8 Supportive Care Ranson > 3 Conservative management < 30% Necrosis CECT > 30% Necrosis Antibiotics Improve No improvement PC drainage

Case Study # 2 32 yr old female PMHx of UTI on Cephalexin X3 d G5P4 38 wks gest. cc: epigastric pain X 3 days with radiation to back & N/V x 10; bilious and non bloody U/A protein 300 mg/dl

Case Study # 2 121 93 5 84 3.1 14 0.1 LIPASE 1330 Amylase 1617 Lactate 2.3 T. B 0.7 AST 14 Alk Phos 92 ALT 13 11.7 12.2 178 36.7 MCV: 89 Bands: 8

Case Study # 2 U/S: CBD 6.2 mm Sludge and mild wall thickening Anything to add to the work up? Triglycerides Trigs: 12,570

Case Study # 2 Emergent C-section done and remained intubated Pink milky intraperitoneal fluid seen during surgery Septic with APACHE score of 15 with 11% risk of mortality Quintin Catheter placed

Case Study #2 CT with Contrast: Severe pancreatitis with no necrosis or pseudocyst or vascular thrombus. Mod ascities. Biliary sludge. No ductal dilatation Plasmapharesis Trigs 471 (10426) Drug Therapy Gemfibrozil 145 mg PO daily

Acute Pancreatitis - Etiology Metabolic Causes Hypertriglyceridemia Pathogenesis Occurs in 1.3 to 3.8 cases of pancreatitis Serum levels >1000 mg/dl Acquired in obesity, DM, hypothyroidism, nephrotic syndrome, estrogen therapy & glucocorticoid excess www.uptodate.com