Management of Head Trauma. Kelley A. Sookraj, MD Kings County Hospital Center June 3rd 2010

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Management of Head Trauma Kelley A. Sookraj, MD Kings County Hospital Center June 3rd 2010

Case Presentation CC: s/p fall HPI: This is an 87 y/o female who fell while walking to the bathroom and hit her head. When seen in ED the patient was found to be AAOx3 with a GCS of 15. She had a c/o headache along with vomiting and she denied any LOC.

PMHx: HTN, Arthritis, CAD, hypercholesterolemia PSHx: CABG 00, stent 03, 06 Allergies: NKDA Meds: lopressor, losartan, aspirin, plavix, sosuvastatin SHx: non-contributory

Vitals: Temp 97.3 F BP 118/48 HR 77 RR 14 O2 sat 100% Physical Exam: General: AAOx3 HEENT: NCAT, EOMI, PERRL C-spine: collar in place, no cervical spine tenderness Chest: CTA bilaterally CVS: S1S2, rrr Abdomen: soft, +BS, NT, ND Back: no TLS tenderness or step-offs Extr: no edema or calf tenderness, FROM Rectal: good tone, no gross blood

Labs: CBC: 8.4 / 8.8 / 27 / 290 Chem: 133 / 3.6 / 95 / 28 / 19 / 0.8 / 120 Coags: 11.4 / 23.3 / 0.8 ABG: 7.483 / 29.2 / 188 / 100% / 23.8 / -1.3 Lactic acid: 1.0 Trop I <0.02

Radiologic Studies: CXR: no rib fractures, no PTX PXR: no fractures Head CT: acute left hemispheric subdural hematoma, 15mm left to right midline shift C-spine CT: no acute fracture, subluxation or prevertebral swelling CT Abd/Pelvis: no intra-abdominal free air or fluid, no evidence of retroperitoneal bleed

Radiologic Studies: Head CT

Consult: Neurosurgery Plan: Admitted to NeuroSurgical ICU NPO, IVF seizure prophylaxis transfusion of platelets Pre-op for OR

Intra-op: Prior to OR the patient became drowsy and was intubated in ED. She was taken to the OR and underwent a left craniotomy with evacuation of subdural hematoma Post-op course: A repeat Head CT was obtained which revealed a re-accumulation of a left hemispheric subdural hematoma with left to right midline shift

Post-op course: Head CT

The patient was taken back to the OR for re-exploration. A large subdural hematoma was found and removed. There was also bleeding from two ruptured anterior bridging veins. Hemostasis was achieved and 2 7mm flat JP drains were left in place. Repeat Head CT showed significant reduction of hematoma and midline shift

Post-op course: Head CT

NSICU course: POD 5: JP drains were removed Head CT showed stabilization of residual hematoma and resolving pneumocephalus She underwent several weaning trials which were unsuccessful and subsequently had a tracheostomy as well as insertion of an IVC filter and PEG tube placement. She was then placed at xxnh.

Management of Head Trauma Glasgow Coma Scale (GCS): neurologic exam that indicates the severity of injury and measures changes in consciousness. EYE-OPENING RESPONSE VERBAL RESPONSE MOTOR RESPONSE Score Response Score Response Score Response 4 spontaneous 5 oriented 6 obeys commands 3 speech 4 confused 5 localizes to pain 2 pain 3 inappropriate 4 withdraws to pain 1 no response 2 incomprehensible 3 flexion to pain 1 no response 2 extension to pain 1 no response

Management of Head Trauma Types of Head Bleed Epidural hematoma - arterial bleed: middle meningeal artery - patients may initially have LOC lucid period sudden deterioration in neurologic status - Head CT: lenticular deformity - operative intervention for significant neurologic deterioration or mass effect

Management of Head Trauma Subdural Hematoma - most common type, usually from tearing of bridging veins between dura and arachnoid - Head CT: crescent shaped deformity - operative intervention for significant mass effect - chronic SDH seen in elderly patients days to week after fall may have focal neurologic deficit, seizures if symptomatic may require burr hole drainage

Management of Head Trauma Subarachnoid Hemorrhage - usually due to cerebral aneurysms (50% from MCA) and/or AVM - severe headache, neurologic deficits, nuchal rigidity - OR for isolation of aneurysm from systemic circulation via clipping

Management of Head Trauma Intracranial Pressure normal 10mmHg, if >20mmHg requires treatment monitoring is indicated if serial neurologic exam is not reliable of worsening intracranial pathology corresponds to a GCS of 8 maximum brain swelling occurs 48-72 hours after injury symptoms: stupor, headache, nausea, vomiting Cushing s triad: HTN, bradycardia, slow RR

Management of Head Trauma CPP = MAP - ICP, maintain between 60-70mmHg Treatment: - ABCs - raise head of bed 30 - relative hyperventilation (CO 2 30-35mmHg) - mannitol load 1g/kg, 0.25mg/kg Q4H - sedation

Management of Head Trauma Surgical Intervention: - extra ventricular drain (EVD): placed within lateral ventricle to drain CSF and measure ICP - craniotomy: burr holes are drilled via skull to remove hematoma and decrease ICP - decompressive craniectomy: part of skull is removed to allow dura mater to expand and allow brain to swell in order to prevent continued crushing parenchymal injury and/or herniation

Management of Head Trauma The Effects of Clopidogrel on Elderly Traumatic Brain Injured Patients Wong, DK et al., Journal of Trauma. 2008; vol 65, pgs 1303-08 Retrospective review of trauma data registry from 2001-2005 www.downstatesurgery.org main outcome: mortality, ICU duration, disposition 131 patients: clopidogrel 21, ASA 90, coumadin 20 Mechanism: fall, MVA, pedestrain struck GCS upon presentation: 14

Management of Head Trauma The Effects of Clopidogrel on Elderly Traumatic Brain Injured Patients Wong, DK et al., Journal of Trauma. 2008; vol 65, pgs 1303-08 Head CT: SDH, majority occurring with pts on ASA, SAH elevated INR for pts on coumadin, transfused FFP 5 pts required craniotomy (1, 2, 2) pts on coumadin- increased ICU stay (10days) pts on clopidogrel- increased mortality rate: 14%, and placement to LT facility: 29%

Management of Head Trauma The Effects of Clopidogrel on Elderly Traumatic Brain Injured Patients Wong, DK et al., Journal of Trauma. 2008; vol 65, pgs 1303-08 Conclusion: patients on clopidogrel have increased longterm disability and fatal consequences when compared to patients on other forms of anticoagulation