HPV DNA Testing. The digene HPV Test. Clinical vs. Analytical Sensitivity. Sample & Assay Technologies

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HPV DNA Testing The digene HPV Test Clinical vs. Analytical Sensitivity Sample & Assay Technologies

How to validate an HPV test for optimal clinical performance Clinical sensitivity more accurately represents an assay s ability to diagnose cervical disease While analytical sensitivity is decisive for HIV or hepatitis C tests, it is much less significant in evaluating a test for the detection of HPV DNA. Testing platforms with high analytical sensitivity (e.g., -based tests) may yield positive results which are clinically irrelevant and, thus, misleading. Number of copies of virus DNA 10 7 10 6 10 5 10 4 10 3 Lesions No lesions Progression to severe lesions Regression of lesions assay cutoff 10 2 10 Normal cytology Abnormal cytology Figure 1. Clinical relevance of a cutoff value in relation to progression risk for CIN+. Adapted from Snijders P.J.F. et al. (1). The thick dashed lines indicate the informative viral load thresholds. Number of copies of virus DNA 10,000,000 1,000,000 100,000 10,000 5,000 1,000 100 10 0 The analytical sensitivity of the test is around 5,000 copies of HPV DNA detection The analytical sensitivity of can correspond to <10 copies of HPV DNA detection Increasing probability of a high-risk HPV induced illness assay cutoff Increasing probability of clinically irrelevant HPV infection Figure 2. Significance of the assay cutoff. This figure compares the clinical relevance of the digene HPV DNA Test and -based methods. The assay cutoff value differentiates clinically irrelevant HPV infections from those that are far more likely to lead to high-risk HPV-induced disease (1). For HPV testing, clinical sensitivity is considerably more important: the goal is to identify as positive only high-risk HPV infections that actually present a likelihood of developing cervical cancer. The test procedure must define a validated threshold value differentiating clinically relevant HPV infections from an HPV DNA presence that does not correlate to future cervical disease (Figure 1). The digene HPV DNA Test (based on Hybrid Capture 2 technology) is the only HPV test procedure enabling physicians to estimate the clinical relevance of high-risk HPV infection using a validated threshold (cutoff) value specifying the number of DNA copies above which a test is regarded as positive. The digene HPV DNA Test s cutoff is approximately 5,000 copies of HPV DNA. To date, all methods lack this threshold value, which is critical for patient management (Figure 2). 2 www.qiagen.com HPV DNA Testing: Clinical vs. Analytical Sensitivity 03/2013

The digene HPV DNA Test is the most extensively validated standardized HPV test Studies involving almost one million patients (2, 3, 4, 5) have been conducted worldwide using the digene HPV DNA Test. Additionally, numerous studies have employed a range of designs and clinical end-points to determine sensitivity in detecting clinically relevant infections with high-risk HPV including dysplasia, pre-cancerous stages, and cervical cancer. Twenty-six selected studies demonstrate that results obtained with the digene HPV DNA Test are superior in clinical sensitivity when compared to methods (Figure 3). Median: 94% Median: 82% 100 90 Clinical sensitivity (%) 80 70 60 50 40 Figure 3. Results, per study, of vs. clinical sensitivity. This figure shows the results of each study s measurement of the digene HPV DNA Test and methods in recognizing pre-cancerous stages (CIN2+). See reference 5. These studies, in total, found that the digene HPV DNA Test yielded results with a median of 94% clinical sensitivity. In contrast, procedures showed a clinical sensitivity of only 82%. The digene HPV DNA Test consistently provided superior sensitivity in detecting pre-cancerous stages required for both primary screening and triage risk assessment (Figure 4). 100 94% 100% 95% Clinical sensitivity (%) 80 60 40 20 0 68% 82% 59% Figure 4. Summary of vs. clinical sensitivity. Numbers not boldfaced indicate highest and lowest sensitivities found in the studies (6). HPV DNA Testing: Clinical vs. Analytical Sensitivity 03/2013 Sample & Assay Technologies 3

The digene HPV DNA Test detects the presence of 13 high-risk HPV types using full-length RNA probes complementary to the HPV DNA, special antibodies, detection reagents, and chemiluminescence. Sample collection is similar to a Pap test, taking a smear of cells from the cervix. The cells in the sample are denatured and the HPV DNA is split into single strands. The basic assay steps are outlined below. Hybridize RNA probe with target DNA. Target DNA combines with specific RNA probes, creating RNA:DNA hybrids. Hybrid capture. RNA:DNA hybrids are captured onto a solid phase coated with universal capture antibodies specific for RNA:DNA hybrids. Signal amplification. Captured RNA:DNA hybrids are detected with multiple antibodies conjugated to alkaline phosphatase. The signal resulting from the chemiluminescent reaction is read and results interpreted. Signal strength is proportional to the HPV load in the sample. The clinically validated cutoff value of the digene HPV DNA Test is 1 pg/ml or approximately 5,000 viral copies/ sample. Samples with lower numbers of virus copies are classified as negative. The digene HPV DNA Test provides the highest clinical sensitivity required for the diagnosis of HPV and patient management. In contrast, in, primers bind to and amplify defined areas of virus DNA presenting a number of disadvantages (Table 1). Due to the absence of a threshold value for -based tests, the number of clinically irrelevant positive results may increase. Additionally, and critically, can signal false negatives in the presence of advanced disease (7). Most methods are based on primers targeting L1 or E1 regions of the HPV genome. These targeted segments may be eliminated when virus DNA is integrated into the human genome a precursor for many advanced stages of the disease. Many methods yield false-negative results that do not recognize a proportion of disease stages such as severe dysplasia and cervical carcinoma. The digene HPV Test The digene HPV DNA Test is a primary tool for detecting only clinically relevant HPV infections The determination of whether a high-risk HPV infection may lead to cervical cancer is highly dependent on the number of detected copies of virus DNA. Only the digene HPV DNA Test provides these clinically significant results, and remains effective throughout all stages of disease progress. HPV DNA Testing: Clinical vs. Analytical Sensitivity 03/2013 Sample & Assay Technologies 4

Chaptertitle X.XX Table 1. and : point-by-point comparison Clinical performance 94% clinical sensitivity 82% clinical sensitivity Standardized method Yes No Risk of false negatives due to inhibition No Yes Risk of false negatives due to L1 or E1 gene deletions No Yes Throughput Up to 264 tests in 8 hours Depends on method Data used to support cervical cancer guidelines Yes No High-risk type profile Currently accepted clinically relevant types 16,18,31,33,35,39,45, 51,52,56,58,59,68 (8) CE-marked and FDA-approved Yes Yes Depends on method Ordering Information Product Contents Cat. no. digene HPV DNA Test 96 tests for 40 cervical samples* 5196-1330 digene High-Risk HPV DNA Test 96 tests for 88 cervical samples 5197-1330 * Includes probe diluent, high-risk and low-risk probes, quality controls, calibrator, capture microplate, reagents, and buffers. Includes probe diluent, high-risk probes, quality controls, calibrator, capture microplate, reagents, and buffers. The digene HPV DNA Test and digene High-Risk HPV DNA Test are intended for in vitro diagnostic use. Visit www.qiagen.com/hpv for further information. For up-to-date licensing information and product-specific disclaimers, see the respective QIAGEN kit handbook or user manual. QIAGEN kit handbooks and user manuals are available at www.qiagen.com or can be requested from QIAGEN Technical Services or your local distributor. Trademarks: QIAGEN, digene, Hybrid Capture (QIAGEN Group). 1075370 03/2013 2013 QIAGEN, all rights reserved. References 1. Snijders, P.J.F., van den Brule, A.J.C., and Meijer, C.J.L.M. (2003) The clinical relevance of human papillomavirus testing: relationship between analytical and clinical sensitivity. J. Pathol. 201, 1. 2. Lorincz, A.T. (2006) HPV testing by hybrid capture. In: Monsonego, J., ed. Emerging Issues on HPV Infections: From Science to Practice. Basel: Karger, p 54. 3. Fetterman, B., Shaber, R., Pawlick, G., and Kinney, W. (2005) Lessons from practice: the first hundred thousand Pap and HPV cotests for general population screening. Poster presented at the 22nd International Papillomavirus Conference and Clinical Workshop. Vancouver. 4. Ronco, G. et al. (2008) Results at recruitment from a randomized controlled trial comparing human papillomavirus testing alone with conventional cytology as the primary cervical cancer screening test. J. Natl. Cancer Inst. 100, 492. 5. Sankaranarayanan, R., et al (2009) HPV Screening for Cervical Cancer in Rural India. N. Engl. J. Med. 360, 1385 1394 6. Lorincz, A.T. and Smith, J.S. (2006) Sexually transmissible viral pathogens: human papillomaviruses and herpes simplex viruses. In: Lorincz, A.T., ed. Nucleic Acid Testing for Human Disease. Boca Raton: Taylor & Francis Group/CRC Press, p 243. 7. Morris, B.J. (2005) Cervical human papillomavirus screening by : advantages of targeting the E6/E7 region. Clin. Chem. Lab. Med. 43, 1171. 8. Wright, T.C. Jr. and Schiffman, M. (2003) Adding a test for human papillomavirus DNA to cervical-cancer screening. N. Engl. J. Med. 348, 489. www.qiagen.com HPV DNA Testing: Clinical vs. Analytical Sensitivity 03/2013

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