Treatment of Hepatitis C in Patients with Renal Insufficiency



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HEPATITIS WEB STUDY HEPATITIS C ONLINE Treatment of Hepatitis C in Patients with Renal Insufficiency Robert G. Gish MD Professor Consultant, Stanford University Medical Center Senior Medical Director, St Josephs Hospital and Medical Center, Liver Program, Phoenix, Arizona (Adjunct) Clinical Professor of Medicine, University of Nevada, Las Vegas Vice-Chair Executive Committee, and Steering Committee Member, National Viral Hepatitis Roundtable Last Updated: July 6, 2015

Robert Gish, MD: Relevant Disclosures To HCV Consulting Board: Bristol-Myers Squibb, Gilead, Merck & Co., Janssen, Abbvie Honoraria for Promotional Talks: Bristol-Myers Squibb, Gilead Sciences, Merck & Co., AbbVie, Janssen

Treatment of Hepatitis C in Patients with Renal Insufficiency Background and Staging of Renal Disease Treatment with Interferon-Based Regimens Treatment with Direct-Acting Antiviral Agents Hepatitis C and Renal Transplantation Summary

TREATMENT OF HCV IN PATIENTS WITH RENAL INSUFFICIENCY Background and Staging Renal Disease

Hepatitis C Treatment Issues Related to Renal Disease Hepatitis C may be associated with or cause renal disease Treatment of hepatitis C and renal disease (1) Treatment in patients with chronic renal insufficiency (2) Treatment to prevent HCV causing renal disease (3) Treatment post renal transplant for renal function and graft survival

Epidemiology of HCV in Patients on Hemodialysis (HD) In US, estimated HCV prevalence of 8% - (approximately 400,000 persons on HD) HCV prevalence 5X greater in HD patients than in general US population Risk factors for HCV infection among hemodialysis patients: - Number of years on dialysis - Number of blood product transfusions - Injection drug use - History of organ transplantation Source: Finelli L, et al. Semi Dial. 2005;18:52-61.

Natural History of HCV Infection in Hemodialysis Patients Impact of Hepatitis C Infection on Hemodialysis Patients: Increased overall risk of mortality Increased risk of cirrhosis Increased incidence of hepatocellular cancer Source: Fabrizi F, et al. J Viral Hepat. 2007;14:697-703.

Hepatitis C and Renal Disease Hepatitis C as a Cause of Renal Disease HCV infection in patients with advanced liver failure increases risk for renal disease Chronic HCV infection associated with increased risk for renal cell carcinoma Chronic HCV infection accelerated renal disease in HIV-infected patients Source: (1) Ozkok A, et al. Gastroenterol. 2014;20:7544-54. (2) Gordon SC, et al. Cancer Epidemiol Biomarkers Prev. 2010;19:1066-73. (3) Peters L, et al. AIDS. 2012;26:1917-26.

Hepatitis C and Renal Disease HCV as a Cause of Renal Disease: Immune Complex Disorders HCV-associated immune complex disorders that cause renal disease Mixed Cryoglobulinemia: + RF as a screening test; reflex to qualitative or quantitative cryoglobulin (type II cryoglobulins) Glomerulonephritis (Membranoproliferative [MPGN] is the most common) Polyarteritis nodosa Uncommon HCV-associated immune complex disorders that cause renal disease Focal segmental glomerular sclerosis Proliferative glomerulonephritis Membranous glomerulonephritis Fibrillary and immunotactoid glomerulopathies Source: (1) Ozkok A, et al. Gastroenterol. 2014;20:7544-54. (2) Gordon SC, et al. Cancer Epidemiol Biomarkers Prev. 2010;19:1066-73. (3) Peters L, et al. AIDS. 2012;26:1917-26.

Stages of Chronic Kidney Disease CKD Stage Description GFR (ml/min/1.73 m 2 ) 1 Kidney Damage with Normal or GFR >90 2 Kidney Damage with Mild GFR 60-89 3 Moderate GFR 30-59 4 Severe GFR 15-29 5 Kidney Failure <15 (or dialysis) Source: NKF KDOQI Clinical Practice Guidelines for Chronic Kidney Disease

TREATMENT OF HCV IN PATIENTS WITH RENAL INSUFFICIENCY Experience with Interferon-Based Therapies

SVR (%) Interferon Monotherapy for HD Patients with Chronic HCV Analysis of the Literature on Efficacy (SVR) 80 70 Analysis of 8 Studies Using INF-alfa 2b Monotherapy 3 million units 3x/week 68 60 50 56 58 40 30 33 27 20 20 21 20 19 10 0 Source: Russo MW, et al. Am J Gastroenterol. 2003;98:1610-5.

Summary Estimates for SVR Rates Peginterferon + Ribavirin for HCV in Hemodialysis Patients Meta-Analysis of the Literature on Efficacy 100 Analysis of 11 Studies (287 patients) Using PEG alfa-2a/peg alfa-2b + RBV 80 86 60 60 54 57 63 40 20 0 All Studies Cohort Studies Controlled Studies Peg-IFN alfa-2a Peg-IFN alfa-2b Source: Fabrizi F, et al. J Viral Hepat. 2014;21:314-24.

PEG-IFN +/- Low-dose RBV (200 mg/day) in HCV GT1 on Hemodialysis HELPER-1 Trial: Study Regimens Week 0 Virologic Responses 48 72 N = 103 Peginterferon alfa-2a + Ribavirin SVR24 N = 94 N = 102 Peginterferon alfa-2a SVR24 N = 91 Drug Dosing Peginterferon alfa-2a 135 µg 1x/week Low-dose Ribavirin: 200 mg once daily Source: Liu CH, et al. Ann Intern Med. 2013;159:729-38.

Virologic Response (%) PEG-IFN +/- Low-dose RBV (200 mg/day) in HCV GT1 on Hemodialysis HELPER-1 Trial: Results Virologic Responses 100 80 87 84 Peginterferon + Ribavirin Peginterferon 60 51 64 40 36 33 20 0 53/103 37/102 90/103 86/102 66/103 34/102 RVR ETVR SVR24 Drug Dosing Peginterferon alfa-2a: 135 µg once weekly Ribavirin: 200 mg daily Source: Liu CH, et al. Ann Intern Med. 2013;159:729-38.

Controversies with Ribavirin Use in Advanced Renal Disease Not recommended with egfr < 50 ml/min/1.73 m 2 in: - Package inserts for Rebetol, Ribasphere - KDIGO 2008 guidelines - 2009 AASLD guidelines Permitted with egfr < 50 ml/min/1.73 m 2 (with dose reduction) in: - Package insert for CoPegus - 2014 AASLD/IDSA/IAS-USA guidelines

TREATMENT OF HCV IN PATIENTS WITH RENAL INSUFFICIENCY Experience with Direct-Acting Antiviral Agents

Treatment of Hepatitis C in Patients with Renal Disease Possible Options using Direct Acting Antiviral Agents Sofosbuvir plus Ribavirin Simeprevir plus Sofosbuvir Ombitasvir-Paritaprevir-Ritonavir plus Dasabuvir (genotype 1) Ledipasvir-Sofosbuvir (pangenotypic) Sofosbuvir plus Daclatasvir* *Daclatasvir was not FDA approved in United States as of July 1, 2015

Sofosbuvir Pharmacokinetics HCV-Negative Patients with Renal Impairment Sofosbuvir Pharmacokinetics in HCV-Negative Patients with Renal Impairment Patient Renal Impairment Sofosbuvir AUC* GS-3310007 AUC* Following Single 400 mg dose of sofosbuvir egfr 50 and < 80 ml/min/1.73 m 2 61% 55% egfr 30 and < 50 ml/min/1.73 m 2 107% 88% egfr <30 ml/min/1.73 m 2 171% 451% ESRD requiring hemodialysis Dosed 1 hour before hemodialysis Dosed 1 hour after hemodialysis *AUC given relative to subjects with normal renal function 28% 60% 1280% 2070% Source: Sofosbuvir Prescribing Information, Gilead Sciences.

Simeprevir Plasma Concentration, ng/ml Simeprevir Pharmacokinetics Severe Renal Impairment versus Healthy Subjects Linear Mean Plasma Concentration-Time Profiles 9,000 Subjects with severe renal impairment (n=8) Matched healthy subjects (n=8) 8,000 7,000 6,000 5,000 4,000 3,000 2,000 1,000 Bars represent standard deviation Source: Janssen Products 0 0 4 8 12 16 20 24 Time, Hours

Sofosbuvir-Containing Regimens including Patients with Renal Disease HCV-TARGET Trial: Study Features HCV-Target and Patients with Renal Disease: Features Design: Longitudinal, cohort study with sofosbuvir-containing regimens, including patients with renal disease Setting: 56 centers in US, Germany, and Canada Entry Criteria - Chronic HCV treated with sofosbuvir-containing regimen - HCV genotype 1-6 - Age 18 or older - Treatment naïve and treatment experienced - Includes patients with baseline renal insufficiency - Includes patients with cirrhosis Primary End-Points - Efficacy (SVR12), safety Source: Saxena V, et al. 50 th EASL. 2015; Abstract LP08.

Patients with SVR 12 (%) Sofosbuvir-Containing Regimens in Patients with Renal Disease HCV -TARGET 100 HCV TARGET: SVR12, by Baseline egfr 80 88 81 89 81 60 40 20 0 15/17 39/48 125/140 1128/1393 30 31-45 46-60 > 60 Estimated Glomerular Filtration Rate (egfr) Source: Saxena V, et al. 50 th EASL. 2015; Abstract LP08.

Patients with SVR 12 (%) Sofosbuvir-Containing Regimens including Patients with Renal Disease HCV-TARGET Trial: Result HCV-TARGET Trial: SVR12 Results by Baseline egfr and Regimen 100 100 100 SOF/PEG/RBV SOF/RBV SOF/SMV SOF/SMV/RBV 100 100 80 80 80 80 93 84 91 92 81 73 87 79 60 40 20 0 33 1/1 4/4 8/10 2/2 1/3 8/10 20/25 9/9 13/14 38/45 62/68 12/13 30 30-45 46-60 >60 Estimated GFR ml/min/1.73 m 2 188/ 232 292/ 400 480/ 552 135/ 171 Abbreviations: SOF = sofosbuvir; PEG = peginterferon; RBV = ribavirin; SMV = simeprevir Source: Saxena V, et al. 50 th EASL. 2015; Abstract LP08.

Ombitasvir-Paritaprevir-Ritonavir and Dasabuvir in GT1 & Renal Disease RUBY-I: Study Design RUBY-I: Features Design: Phase 3b, randomized, open-label trial evaluating safety and efficacy of 3D (ombitasvir-paritaprevir-ritonavir and dasabuvir) with or without ribavirin for 12 weeks in treatment-naïve patients with chronic HCV GT1 and advanced kidney disease Setting: 9 sites in United States Entry Criteria - Adults with chronic HCV genotype 1 infection - Chronic kidney disease stage 4 or 5 (egfr <30 ml/min/1.73 m 2 ) +/- HD - Plasma HCV RNA greater than 1,000 IU/mL - Absence of cirrhosis - Absence of coinfection with HBV or HIV - Baseline Hb 10 g/dl Primary End-Point: SVR12 Source: Pockros PJ, et al. 50 th EASL. 2015; Abstract L01.

Ombitasvir-Paritaprevir-Ritonavir and Dasabuvir in GT1 & Renal Disease RUBY-I: Regimens Week 0 12 24 GT 1a n = 13 Ombitasvir-Paritaprevir-Ritonavir and Dasabuvir + Ribavirin SVR12 GT 1b n = 7 Ombitasvir-Paritaprevir-Ritonavir and Dasabuvir SVR12 Drug Dosing Ombitasvir-Paritaprevir-Ritonavir (25/150/100 mg once daily) + Dasabuvir: 250 mg twice daily Ribavirin for patients not on hemodialysis: 200 mg once daily Ribavirin for patients on hemodialysis: 200 mg given 4 hours before each hemodialysis session Source: Pockros PJ, et al. 50 th EASL. 2015; Abstract L01.

Virologic Response Ombitasvir-Paritaprevir-Ritonavir and Dasabuvir in GT1 & Renal Disease RUBY-I: Baseline Results RUBY-I: SVR 12 Rates* 100 GT1a: 3D + RBV 100 100 100 100 100 GT1b: 3D 80 60 40 20 0 12/12 2/2 85/88 9/9 1/1 91/91 2/2 0/0 EOT SVR4 SVR12 3D = Ombitasvir-Paritaprevir-Ritonavir and Dasabuvir; RBV = ribavirin; EOT = end of treatment Source: Pockros PJ, et al. 50 th EASL. 2015; Abstract L01.

AASLD/IDSA/IAS-USA 2015 HCV Treatment Recommendations Recommendations for Patients with Renal Impairment AASLD/IDSA Recommendations for Patients with Renal Impairment* Dosage adjustments for patients with mild to moderate renal impairment (CrCl 30 ml/min-80 ml/min) Sofosbuvir: no dosage adjustment required Simeprevir: no dosage adjustment required Ledipasvir-sofosbuvir: no dosage adjustment required Ombitasvir-paritaprevir-ritonavir + dasabuvir: no dosage adjustment required Dosage adjustments for patients with severe renal impairment (CrCl <30 ml/min or ESRD) Treatment can be contemplated after consultation with an expert, because safety and efficacy data are not available for these patients. *Recommendations for patients with renal impairment, including severe renal impairment (creatinine clearance <30 ml/min) or end-stage renal disease requiring hemodialysis or peritoneal dialysis Source: AASLD/IDSA/IAS-USA (www.hcvguidelines.org). Viewed June 26, 2015

TREATMENT OF HCV IN PATIENTS WITH RENAL INSUFFICIENCY HCV and Renal Transplantation

Impact of HCV on Outcome of Renal Transplantation HCV increases glomerulonephritis in transplanted kidney HCV reduces renal allograft survival HCV decreases long-term patient survival HCV infection is not a contraindication to renal transplantation unless portal hypertension is present or there is decompensated liver disease since patient survival with RT is better than with dialysis Source: Baid-Agrawal S, et al. Am J Transplant. 2014. August [Epub ahead of print]

Relative Risk of Death Relative Risk of Death among Patients Undergoing Renal Transplantation versus those who Remained on Dialysis Relative Risk of Death (all causes): Transplanted versus Dialysis 6 5 4 4.75 Above red line = higher death risk with Renal Transplant Below red line = higher death risk with Dialysis 3 2 1 0 1.76 0.84 0.31 0 to 3 months 3 to 6 months 7 months to 4 years Longer than 4 years Source: Pereira BJG, et al. Kidney Int. 1998;53:1374-81.

Hepatitis C and Renal Disease Rationale for HCV Treatment in Renal Transplant Candidate Eradicate HCV as immunologic stimulus to B-cells to decrease immune complex formation and impact vasculitis or glomerulonephritis Decrease extrahepatic HCV-related complications Prevent HCV-related post-transplant complications - Interaction with HCV immune complexes and calcineurin inhibitor related renal toxicity HCV-related liver disease may accelerate with post-transplant immunosuppression Post-transplant treatment extremely difficult due to risk of graft rejection from interferon (historical)

Treatment of HCV after Renal Transplantation Interferon-based therapy relatively contraindicated because of risk of allograft rejection and loss Post-transplant interferon/ribavirin recommended only for - Fibrosing cholestatic hepatitis IF daclatasvir compassionate use not available - Life-threatening vasculitis Interferon-free regimens will provide new options

Treatment of HCV Post-Renal Transplant Renal function less problematic depending on: - Use, dose, & blood levels of calcineurin inhibitor (cyclosporine, tacrolimus) - Improvement in GFR with graft recovery - History of rejection and residual renal damage Address drug-drug interactions per medication & drug class Higher HCV RNA levels due to immunosuppression may impact SVR rates No effective therapy yet published in controlled trials

TREATMENT OF HCV IN PATIENTS WITH RENAL INSUFFICIENCY Summary and Recommendations

Treatment of Hepatitis C in Patients with Renal Insufficiency Summary Points Renal disease severity should guide treatment decisions Interferon- and Peginterferon-based Rx of historical importance only Maximize EPO use when using ribavirin in this patient population First-generation HCV protease inhibitors not recommended No dose adjustments with DAAs if GFR 30 ml/min Limited data with DAAs in patients with GFR <30 ml/min Obtain expert consultation if GFR <30 ml/min, especially HD patients Renal transplant candidates should receive HCV treatment with DAAs - Either before or after transplantation, depending on clinical scenario