Chapter 11. Training Adaptations. Text Sources 1. Nelson Physical Education VCE Units 3&4: 4 th Edition Malpeli, Horton, Davey and Telford 2006.

Transcription

1 Chapter 11 Training Adaptations Text Sources 1. Nelson Physical Education VCE Units 3&4: 4 th Edition Malpeli, Horton, Davey and Telford Live It Up 2: 2 nd Edition Smyth, Brown, Judge, McCallum and Pritchard 2006.

2 Principle of Adaptation Training Adaptations

3 Principle of Adaptation Athletes train to adapt their bodies to a particular sport/activity. Training should be; Specific to their sport Specific to the desired outcome as a result of adaptations. SAID Principle S = Specific A = Adaptation I = Imposed D = Demands Adaptation = a long-term physiological change in response to training loads that allows the body to meet new demands. Stress on the body causes adaptations. A plateau occurs when the training load is not sufficient to cause stress. Adaptations can be classified as acute and chronic; Acute Immediate physiological response to exercise which last the duration of the exercise session. Type of training not important. Chronic Long-term adaptations to exercise. In this chapter, we will focus on chronic changes.

4 Anaerobic and Aerobic Adaptations Training Adaptations

5 Anaerobic Energy System Adaptations Anaerobic Training the ATP-PC and lactic acid systems cause; Increased levels of anaerobic enzymes and fuels (See fig p.263) Increase in glycolytic capacity Improvements at the muscular level in both systems Aerobic Improvements in; Oxygen uptake Transport and utilisation of oxygen Fat breakdown as a fuel Fatty acid oxidation and respiratory ATP production Lactate Infection Point (See fig 11.5 and 11.6 p.265) Reduced carbohydrate use during sub-maximal exercise Increased use of blood glucose assisting in glycogen sparing. Increased capillarisation, mitochondria density and oxidative enzymes.

6 Checkpoints Complete questions 1-6 page 265 of Nelson Physical Education VCE Units 3 & 4.

7 Cardiovascular Training Adaptations Training Adaptations

8 Cardiovascular Training Cardiac Hypertrophy - Heart increases (Left ventricle) in size as a result of training. This increases the stroke volume (SV) Increased capillarisation - (Coronary blood supply) of the heart increases blood flow to the heart. Increased stroke volume - thus reducing HR. Lowered resting heart rate (Increase in SV causes a decrease in HR when Q is constant approx 5 litres) Lower heart rate during sub-maximal workloads Due to increased SV. Improved heart-rate recovery rates Due to increased SV Increases cardiac output at maximum workload Constant at rest, but Q can reach up to 30L/min in elite athletes. Cardiac Output = Stroke Volume x Heart Rate Q = SV x HR Example Q = 5L SV = Q/HR Before training HR = 71bpm therefore the SV = 0.07L/beat After training program HR=50bpm. SV now = 0.1L/beat

9 Cardiovascular Training Lower blood pressure relieves hypertension by lowering resistance in the vessels Arterio-venous oxygen difference - increases as athlete is able to use oxygen from arteries more effectively (see fig & p.271) Increased plasma, blood volume and haemoglobin levels Increased capillarisation of skeletal muscle Decreased blood cholesterol, triglycerides ad Low Density Lipoproteins (LDP). These substances are associated with coronary heart disease. Increased high density lipoproteins (HDL) Ratio of HDL to LDL increases, which is important for heart health. Increased redistribution of blood Training can lead to a 20% increase in blood flow to working muscles.

10 Coursework 11.1 Complete the data analysis task on page 267 of Nelson Physical Education VCE Units 3 & 4.

11 Coursework 11.2 Complete the laboratory task on page of Nelson Physical Education VCE Units 3 & 4.

12 Respiratory Training Adaptations Training Adaptations

13 Respiratory Training Adaptations Decreased minute ventilation- Lungs become more efficient as a result of training. Ventilation is therefore reduced at sub-maximal workloads. Increased pulmonary diffusion oxygen is more readily extracted from the alveoli Increased tidal volume (Amount of air inspired and expired during breathing) Ventilatory musculature Muscles responsible for breathing require less oxygen. Improved lung function due to improved lung volume and alveolar capacity surface area. Aerobic capacity Improves due to an increase in oxygen supply to the working muscles. See table 11.4 p.273 Increased VO 2 max Due to; Increase in cardiac output, Increase in RBC numbers, Increase in a-vo 2 diff Increase in muscle capillarisation Improved oxygen extraction.

14 Oxygen extraction: a-v0 2 difference a-v0 2 difference = Arteriovenous oxygen difference: difference in oxygen consumption when comparing that in the arterioles to the venules, and an indirect measure of how much oxygen muscles are using An increase in a-v0 2 difference results in more blood being pumped to active muscles (especially slow-twitch) Muscle fibres better at extracting and processing oxygen as a result of increased mitochondria numbers, more oxidative enzymes and increased levels of myoglobin. All of this is due to the oxygen demands of the muscles 18 ml/100ml 12 ml/100ml

15 Checkpoints Complete questions 1-4 page 273 of Nelson Physical Education VCE Units 3 & 4.

16 Coursework 11.3 Complete the laboratory task on page 274 of Nelson Physical Education VCE Units 3 & 4.

17 Muscular Training Adaptations Training Adaptations

18 Muscular Training Adaptations Athletes need to use specific training methods to cause muscular adaptations for their sport. Aerobic Trains the slow twitch (Type I) fibres. Anaerobic Trains fast twitch (Type II) fibres. Muscle fibre type and percentage that make up the body Muscle fibre type can change, eg for elite endurance athletes from 70-90% Genetics a big advantage to start with x amount of fibre percentage You are born with x amount of fast and slow twitch fibres. BUT you can train and gain more of one type. MYTH with training you can change from fast twitch to slow twitch or vice versa. IMPOSSIBLE HOWEVER fast twitch fibres have been known to take on slow twitch characteristics in response to aerobic training

19 Aerobic (Muscular) Increased oxygen utilisation Increased size and number and density of mitochondria Increased myoglobin stores. Increased muscular fuel stores ie.glycogen, fatty acids, triglycerides and oxidative enzymes. Increased capillary density to slow twitch fibres. Increased use of fat at sub-maximal levels. Increased stores and use of intramuscular triglycerides. Increased oxidation of glucose and fats Ability to metabolise and extract energy has improved. The body can therefore use glycogen sparing. Decreased use of lactic acid system Some muscle fibre adaptation.

20 Anaerobic (Muscular) Muscular Hypertrophy Enlargement of the fast twitch muscle fibres Increased muscular stores of ATP, PC, creatine and glycogen. Increased ATP-PC splitting and resynthesis of enzymes Increased glycolytic capacity Enhances lactic acid systems ability to use glycogen. Cardiac hypertrophy Increases contraction forces exerted by the left ventricle in the heart.

21 Anaerobic (Muscular) Increased contractile proteins in muscles. Increased myosin ATPase Molecule responsible for splitting ATP into ADP Increased muscle buffering capacity Muscles able to tolerate higher levels of fatiguing products Muscle hyperplasia Research in animals suggest that new muscle fibres may form under stress. Other Increase in strength of connective tissue, number of motor units, speed on nerve impulses and muscular contraction speed.

22 Adaptations

23 Coursework 11.4 Complete the case study on page 280 of Nelson Physical Education VCE Units 3 & 4.

24 Adaptations are Reversible Training Adaptations

25 Adaptations are Reversible Adaptations are reduced and then lost after stopping regular training. See table 11.6 p.281 The reversibility principle applies when an athlete becomes inactive. As a result, athletes need to undertake a vigorous pre-season months before the in-season starts. Therefore maintenance in the offseason is required to minimise reversing the adaptations.

26 Checkpoints Complete questions 1-5 page 281 of Nelson Physical Education VCE Units 3 & 4.

27 Test Your Knowledge Complete the review questions 1-5 page 283 of Nelson Physical Education VCE Units 3 & 4.

28 Peak Performance Complete the chapter questions on page of Nelson Peak Performance Physical Education VCE Units 3 & 4.

29 PHYS ED Notes Read the summarised information of pages of PHYS ED Notes and complete the revision questions.

30 VCAA Questions

31 Web Links Chapter 11 Australian Institute of Sport, strength and conditioning: Australian Sports Commission: Find 30 promotion (Government of WA Department of Health): Walking School Bus promotion (UK): Ministry of Health (New Zealand) toolkits: The 10,000 Steps Rockhampton project: Travelsmart Australia: World Health Organisation: Heart Foundation Australia: VicHealth (The Victorian Health Promotion Foundation): Be Active promotion (Government of South Australia): Go For Your Life: Physical Activity Resources for Health Professionals Introduction (Centre for Phys/cardiopulmonary.html disease control and prevention USA): Health Sports Promotion science (Public Health library Agency of Canada): (Gatorade Sports Science Institute): Strategic Inter-Governmental Forum on Physical Activity and Health (SIGPAH): Healthy youth (Centre for disease control and prevention (USA): America On The Move promotion: Papers Sport from science the International (site Journal of for Behavioural sports Nutrition research): and Physical Activity: Department of health and aging (Australian government): Building a healthy, active Australia (Australian government): National Public Health Partnership: Be Active promotion (Government of South Australia): Sport and Recreation Australia: Exercise Physiology The methods and mechanisms underlying performance: Information about metabolic adaptations and cardiovascular physiology: PowerPoint presentation about cardiovascular adaptations from aerobic training (Illinois Wesleyan University USA): How stuff works How exercise works: Article Strength Training Basics: